Recherchemedicale.com
Phénomènes physiologiques cellulaires
Phénomènes physiologiques cellulaires : Questions médicales fréquentes
Termes MeSH sélectionnés :
Diagnostic
5
Fonction cellulaire
Tests de laboratoire
Stress cellulaire
Protéines de choc thermique
Apoptose
Tests de laboratoire
Prolifération cellulaire
Cytométrie en flux
Dysfonction mitochondriale
ATP
Symptômes
5
Vieillissement cellulaire
Dommages à l'ADN
Nécrose
Membrane cellulaire
Apoptose
Fragmentation de l'ADN
Prévention
5
Stress cellulaire
Prévention
Vieillissement cellulaire
Habitudes de vie
Nécrose
Circulation sanguine
Inflammation
Alimentation
Traitements
5
Stress oxydatif
Antioxydants
Dysfonction mitochondriale
Coenzyme Q10
Inflammation
Anti-inflammatoires
Apoptose
Inhibiteurs de caspases
Vieillissement cellulaire
Thérapies régénératives
Complications
5
Stress oxydatif
Maladies cardiovasculaires
Dysfonction mitochondriale
Troubles métaboliques
Inflammation chronique
Maladies auto-immunes
Apoptose
Maladies dégénératives
Vieillissement cellulaire
Maladie d'Alzheimer
Facteurs de risque
5
Stress cellulaire
Facteurs de risque
Vieillissement cellulaire
Génétique
Toxines
Dommages cellulaires
Hypoxie
Maladies respiratoires
Inflammation
Alimentation déséquilibrée
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"name": "Quels tests pour détecter l'apoptose ?",
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"name": "Comment mesurer la prolifération cellulaire ?",
"position": 4,
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"name": "Quels signes indiquent une dysfonction mitochondriale ?",
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"name": "Quels symptômes d'une hypoxie cellulaire ?",
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"name": "Quels signes d'une apoptose active ?",
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"@type": "Question",
"name": "Comment prévenir le stress cellulaire ?",
"position": 11,
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{
"@type": "Question",
"name": "Quelles habitudes pour éviter le vieillissement cellulaire ?",
"position": 12,
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{
"@type": "Question",
"name": "Comment réduire le risque de nécrose cellulaire ?",
"position": 13,
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"name": "Quelles stratégies pour prévenir l'hypoxie ?",
"position": 14,
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"name": "Comment prévenir l'inflammation chronique ?",
"position": 15,
"acceptedAnswer": {
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"text": "Une alimentation riche en oméga-3 et des exercices réguliers peuvent réduire l'inflammation."
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{
"@type": "Question",
"name": "Comment traiter le stress oxydatif ?",
"position": 16,
"acceptedAnswer": {
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"text": "Des antioxydants comme la vitamine C et E peuvent aider à réduire le stress oxydatif."
}
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{
"@type": "Question",
"name": "Quels traitements pour la dysfonction mitochondriale ?",
"position": 17,
"acceptedAnswer": {
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}
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{
"@type": "Question",
"name": "Comment gérer l'inflammation cellulaire ?",
"position": 18,
"acceptedAnswer": {
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"text": "Des anti-inflammatoires non stéroïdiens (AINS) et des corticostéroïdes sont prescrits."
}
},
{
"@type": "Question",
"name": "Quels traitements pour l'apoptose excessive ?",
"position": 19,
"acceptedAnswer": {
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"text": "Des inhibiteurs de caspases peuvent être utilisés pour moduler l'apoptose."
}
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{
"@type": "Question",
"name": "Comment traiter les maladies liées au vieillissement cellulaire ?",
"position": 20,
"acceptedAnswer": {
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{
"@type": "Question",
"name": "Quelles complications du stress oxydatif ?",
"position": 21,
"acceptedAnswer": {
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"text": "Le stress oxydatif peut entraîner des maladies cardiovasculaires et neurodégénératives."
}
},
{
"@type": "Question",
"name": "Quels risques liés à la dysfonction mitochondriale ?",
"position": 22,
"acceptedAnswer": {
"@type": "Answer",
"text": "Elle peut causer des troubles métaboliques et des maladies neurodégénératives."
}
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{
"@type": "Question",
"name": "Quelles complications de l'inflammation chronique ?",
"position": 23,
"acceptedAnswer": {
"@type": "Answer",
"text": "L'inflammation chronique peut mener à des maladies auto-immunes et des cancers."
}
},
{
"@type": "Question",
"name": "Quels effets de l'apoptose excessive ?",
"position": 24,
"acceptedAnswer": {
"@type": "Answer",
"text": "Une apoptose excessive peut contribuer à des maladies dégénératives et au cancer."
}
},
{
"@type": "Question",
"name": "Quelles complications du vieillissement cellulaire ?",
"position": 25,
"acceptedAnswer": {
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"text": "Le vieillissement cellulaire est associé à des maladies comme l'Alzheimer et le diabète."
}
},
{
"@type": "Question",
"name": "Quels facteurs de risque pour le stress cellulaire ?",
"position": 26,
"acceptedAnswer": {
"@type": "Answer",
"text": "Le tabagisme, l'obésité et le manque d'exercice augmentent le stress cellulaire."
}
},
{
"@type": "Question",
"name": "Quels facteurs influencent le vieillissement cellulaire ?",
"position": 27,
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"@type": "Answer",
"text": "La génétique, l'alimentation et l'exposition aux toxines jouent un rôle clé."
}
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{
"@type": "Question",
"name": "Quels risques liés à l'exposition aux toxines ?",
"position": 28,
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{
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"name": "Quels facteurs de risque pour l'hypoxie ?",
"position": 29,
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{
"@type": "Question",
"name": "Quels facteurs de risque pour l'inflammation ?",
"position": 30,
"acceptedAnswer": {
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Expert en Médecine, Optimisation des Parcours de Soins et Révision Médicale
Validation scientifique effectuée le 02/02/2025
Contenu vérifié selon les dernières recommandations médicales
2 publications dans cette catégorie
Affiliations :
Arizona Cancer Evolution Center, Arizona State University, Tempe, AZ, USA. stefania.kapsetaki@tufts.edu.
Biodesign Center for Biocomputing, Security and Society, Arizona State University, Tempe, AZ, USA. stefania.kapsetaki@tufts.edu.
Department of Biology, School of Arts and Sciences, Tufts University, Medford, MA, USA. stefania.kapsetaki@tufts.edu.
Publications dans "Phénomènes physiologiques cellulaires" :
2 publications dans cette catégorie
Affiliations :
Arizona Cancer Evolution Center, Arizona State University, Tempe, AZ, USA.
Biodesign Center for Biocomputing, Security and Society, Arizona State University, Tempe, AZ, USA.
School of Life Sciences, Arizona State University, Tempe, AZ, USA.
Publications dans "Phénomènes physiologiques cellulaires" :
2 publications dans cette catégorie
Affiliations :
Arizona Cancer Evolution Center, Arizona State University, Tempe, AZ, USA.
Biodesign Center for Biocomputing, Security and Society, Arizona State University, Tempe, AZ, USA.
School of Life Sciences, Arizona State University, Tempe, AZ, USA.
Biodesign Center for Mechanisms of Evolution, Arizona State University, Tempe, AZ, USA.
Center for Evolution and Medicine, Arizona State University, Tempe, AZ, USA.
Publications dans "Phénomènes physiologiques cellulaires" :
1 publication dans cette catégorie
Affiliations :
Australian Institute of Microbiology and Infection, University of Technology Sydney, Broadway, New South Wales, Australia.
Publications dans "Phénomènes physiologiques cellulaires" :
1 publication dans cette catégorie
Affiliations :
Australian Institute of Microbiology and Infection, University of Technology Sydney, Broadway, New South Wales, Australia.
Publications dans "Phénomènes physiologiques cellulaires" :
1 publication dans cette catégorie
Affiliations :
Australian Institute of Microbiology and Infection, University of Technology Sydney, Broadway, New South Wales, Australia.
Australian Centre for Genomic Epidemiological Microbiology, University of Technology Sydney, Broadway, New South Wales, Australia.
Publications dans "Phénomènes physiologiques cellulaires" :
1 publication dans cette catégorie
Affiliations :
Australian Institute of Microbiology and Infection, University of Technology Sydney, Broadway, New South Wales, Australia.
Australian Centre for Genomic Epidemiological Microbiology, University of Technology Sydney, Broadway, New South Wales, Australia.
Publications dans "Phénomènes physiologiques cellulaires" :
1 publication dans cette catégorie
Affiliations :
Australian Institute of Microbiology and Infection, University of Technology Sydney, Broadway, New South Wales, Australia.
Publications dans "Phénomènes physiologiques cellulaires" :
1 publication dans cette catégorie
Affiliations :
Australian Institute of Microbiology and Infection, University of Technology Sydney, Broadway, New South Wales, Australia.
School of Chemical Engineering, University of New South Wales, Sydney, New South Wales, Australia.
Publications dans "Phénomènes physiologiques cellulaires" :
1 publication dans cette catégorie
Affiliations :
Division of Regenerative Biology, School of Biomedical Sciences, University of Western Australia, Perth, 6009, Australia.
Publications dans "Phénomènes physiologiques cellulaires" :
1 publication dans cette catégorie
Affiliations :
Division of Regenerative Biology, School of Biomedical Sciences, University of Western Australia, Perth, 6009, Australia.
Publications dans "Phénomènes physiologiques cellulaires" :
1 publication dans cette catégorie
Affiliations :
Guangdong Provincial Key Laboratory of Industrial Surfactant, Guangdong Research Institute of Petrochemical and Fine Chemical Engineering, Guangdong Academy of Sciences, Guangzhou, 510665, China.
Division of Regenerative Biology, School of Biomedical Sciences, University of Western Australia, Perth, 6009, Australia.
Publications dans "Phénomènes physiologiques cellulaires" :
1 publication dans cette catégorie
Affiliations :
Guangdong Provincial Key Laboratory of Industrial Surfactant, Guangdong Research Institute of Petrochemical and Fine Chemical Engineering, Guangdong Academy of Sciences, Guangzhou, 510665, China.
Publications dans "Phénomènes physiologiques cellulaires" :
1 publication dans cette catégorie
Affiliations :
Division of Regenerative Biology, School of Biomedical Sciences, University of Western Australia, Perth, 6009, Australia.
Publications dans "Phénomènes physiologiques cellulaires" :
1 publication dans cette catégorie
Affiliations :
Division of Regenerative Biology, School of Biomedical Sciences, University of Western Australia, Perth, 6009, Australia.
Publications dans "Phénomènes physiologiques cellulaires" :
1 publication dans cette catégorie
Affiliations :
Institute of Cytology, Russian Academy of Sciences, 194064 St. Petersburg, Russia.
Publications dans "Phénomènes physiologiques cellulaires" :
1 publication dans cette catégorie
Affiliations :
Histology and Embryology Students' Science Association, Department of Histology and Embryology, Faculty of Medicine, Warsaw Medical University, Chalubinskiego 5, 02-004 Warsaw, Poland.
Publications dans "Phénomènes physiologiques cellulaires" :
1 publication dans cette catégorie
Affiliations :
Histology and Embryology Students' Science Association, Department of Histology and Embryology, Faculty of Medicine, Warsaw Medical University, Chalubinskiego 5, 02-004 Warsaw, Poland.
Publications dans "Phénomènes physiologiques cellulaires" :
1 publication dans cette catégorie
Affiliations :
Histology and Embryology Students' Science Association, Department of Histology and Embryology, Faculty of Medicine, Warsaw Medical University, Chalubinskiego 5, 02-004 Warsaw, Poland.
Publications dans "Phénomènes physiologiques cellulaires" :
1 publication dans cette catégorie
Affiliations :
Faculty of Medicine, Collegium Medicum, Cardinal Stefan Wyszyński University in Warsaw, Dewajtis 5, 01-815 Warsaw, Poland.
Faculty of Medicine, University of Technology, Rolna 43, 40-555 Katowice, Poland.
Publications dans "Phénomènes physiologiques cellulaires" :
Acinetobacter baumannii is a Gram-negative opportunistic bacterium responsible for nosocomial and community-acquired infections. This pathogen is globally disseminated and associated with high levels ...
Acinetobacter is a gram-negative nosocomial pathogenic bacteria. The contributing factor for the pathogenicity of Acinetobacter is severe due to its property of antibacterial drug resistance. Often an...
Mediastinitis remains one of the most serious complications of cardiac surgery. The reported incidence is 1-4%, while the related mortality varies from 10-47%....
A patient with triple vessel disease (TVD) was hospitalized at our clinic for coronary artery bypass graft (CABG) surgery. The preoperative examination results were normal. We performed standard CABG ...
Mediastinitis is associated with high mortality and high financial and human costs. The occurrence of this high-risk complication can be prevented through constant vigilance at every step from admissi...
Acinetobacter baumannii is an important nosocomial and opportunistic pathogen. It causes infections worldwide, especially in intensive care units. It is clinically significant owing to its ability to ...
The aminoglycoside antibiotics amikacin, gentamicin, and tobramycin are important therapeutic options for Acinetobacter iinfections. Several genes that confer resistance to one or more of these antibi...
Acinetobacter baumannii is a Gram-negative, multidrug-resistant (MDR) pathogen that causes nosocomial infections, especially in intensive care units (ICUs) and immunocompromised patients. A. baumannii...
Among the bacterial species included in the ESKAPE group, Acinetobacter baumannii is of great interest due to its intrinsic and acquired resistance to many antibiotics and its ability to infect differ...
Acinetobacter baumannii is a common pathogen in hospitals and usually causes bacteremia, pneumonia, meningitis, peritonitis and other diseases. Isolates carried NDM-1 gene can make several antibiotics...
Acinetobacter baumannii is a Gram-negative nosocomial pathogen associated with significant disease. Crucial to the survival and pathogenesis of A. baumannii is the ability to acquire essential micronu...
Acinetobacter baumannii is an opportunistic Gram-negative bacterial pathogen that poses a threat for frail patients worldwide. The high ability to withstand environmental stresses as well as its resis...