Titre : Phénomènes physiologiques cellulaires

Phénomènes physiologiques cellulaires : Questions médicales fréquentes

Questions fréquentes et termes MeSH associés

Diagnostic 5

#1

Comment évaluer la fonction cellulaire ?

Des tests de culture cellulaire et des analyses biochimiques sont utilisés.
Fonction cellulaire Tests de laboratoire
#2

Quels marqueurs indiquent un stress cellulaire ?

Des marqueurs comme la protéine HSP70 et les niveaux de ROS sont souvent mesurés.
Stress cellulaire Protéines de choc thermique
#3

Quels tests pour détecter l'apoptose ?

Des tests comme l'Annexin V et la coloration par DAPI sont couramment utilisés.
Apoptose Tests de laboratoire
#4

Comment mesurer la prolifération cellulaire ?

Des méthodes comme le test MTT ou la cytométrie en flux sont efficaces.
Prolifération cellulaire Cytométrie en flux
#5

Quels signes indiquent une dysfonction mitochondriale ?

Une diminution de l'ATP et une augmentation des espèces réactives de l'oxygène sont des indicateurs.
Dysfonction mitochondriale ATP

Symptômes 5

#1

Quels symptômes d'une inflammation cellulaire ?

Rougeur, chaleur, douleur et gonflement sont des signes d'inflammation.
Inflammation Symptômes
#2

Comment reconnaître le vieillissement cellulaire ?

Diminution de la régénération et accumulation de dommages ADN sont des indicateurs.
Vieillissement cellulaire Dommages à l'ADN
#3

Quels signes d'une nécrose cellulaire ?

Les signes incluent la décoloration, la déformation et la rupture de la membrane cellulaire.
Nécrose Membrane cellulaire
#4

Quels symptômes d'une hypoxie cellulaire ?

Fatigue, essoufflement et cyanose peuvent indiquer une hypoxie au niveau cellulaire.
Hypoxie Symptômes
#5

Quels signes d'une apoptose active ?

La fragmentation de l'ADN et la condensation nucléaire sont des signes d'apoptose.
Apoptose Fragmentation de l'ADN

Prévention 5

#1

Comment prévenir le stress cellulaire ?

Une alimentation équilibrée et l'exercice régulier aident à réduire le stress cellulaire.
Stress cellulaire Prévention
#2

Quelles habitudes pour éviter le vieillissement cellulaire ?

Éviter le tabac, limiter l'alcool et pratiquer une activité physique régulière sont bénéfiques.
Vieillissement cellulaire Habitudes de vie
#3

Comment réduire le risque de nécrose cellulaire ?

Maintenir une bonne circulation sanguine et éviter les toxines peut aider.
Nécrose Circulation sanguine
#4

Quelles stratégies pour prévenir l'hypoxie ?

Éviter les environnements à faible oxygène et pratiquer des exercices respiratoires sont conseillés.
Hypoxie Prévention
#5

Comment prévenir l'inflammation chronique ?

Une alimentation riche en oméga-3 et des exercices réguliers peuvent réduire l'inflammation.
Inflammation Alimentation

Traitements 5

#1

Comment traiter le stress oxydatif ?

Des antioxydants comme la vitamine C et E peuvent aider à réduire le stress oxydatif.
Stress oxydatif Antioxydants
#2

Quels traitements pour la dysfonction mitochondriale ?

Des cofacteurs comme le coenzyme Q10 et des thérapies ciblées sont souvent utilisés.
Dysfonction mitochondriale Coenzyme Q10
#3

Comment gérer l'inflammation cellulaire ?

Des anti-inflammatoires non stéroïdiens (AINS) et des corticostéroïdes sont prescrits.
Inflammation Anti-inflammatoires
#4

Quels traitements pour l'apoptose excessive ?

Des inhibiteurs de caspases peuvent être utilisés pour moduler l'apoptose.
Apoptose Inhibiteurs de caspases
#5

Comment traiter les maladies liées au vieillissement cellulaire ?

Des thérapies régénératives et des médicaments ciblant les voies de vieillissement sont explorés.
Vieillissement cellulaire Thérapies régénératives

Complications 5

#1

Quelles complications du stress oxydatif ?

Le stress oxydatif peut entraîner des maladies cardiovasculaires et neurodégénératives.
Stress oxydatif Maladies cardiovasculaires
#2

Quels risques liés à la dysfonction mitochondriale ?

Elle peut causer des troubles métaboliques et des maladies neurodégénératives.
Dysfonction mitochondriale Troubles métaboliques
#3

Quelles complications de l'inflammation chronique ?

L'inflammation chronique peut mener à des maladies auto-immunes et des cancers.
Inflammation chronique Maladies auto-immunes
#4

Quels effets de l'apoptose excessive ?

Une apoptose excessive peut contribuer à des maladies dégénératives et au cancer.
Apoptose Maladies dégénératives
#5

Quelles complications du vieillissement cellulaire ?

Le vieillissement cellulaire est associé à des maladies comme l'Alzheimer et le diabète.
Vieillissement cellulaire Maladie d'Alzheimer

Facteurs de risque 5

#1

Quels facteurs de risque pour le stress cellulaire ?

Le tabagisme, l'obésité et le manque d'exercice augmentent le stress cellulaire.
Stress cellulaire Facteurs de risque
#2

Quels facteurs influencent le vieillissement cellulaire ?

La génétique, l'alimentation et l'exposition aux toxines jouent un rôle clé.
Vieillissement cellulaire Génétique
#3

Quels risques liés à l'exposition aux toxines ?

L'exposition à des produits chimiques peut provoquer des dommages cellulaires et des cancers.
Toxines Dommages cellulaires
#4

Quels facteurs de risque pour l'hypoxie ?

Les maladies respiratoires et les environnements à faible oxygène augmentent le risque d'hypoxie.
Hypoxie Maladies respiratoires
#5

Quels facteurs de risque pour l'inflammation ?

Le stress, l'alimentation déséquilibrée et le manque d'exercice sont des facteurs de risque.
Inflammation Alimentation déséquilibrée
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"https://recherchemedicale.com/mesh/D002468#section-facteurs de risque" } ] }, { "@type": "FAQPage", "mainEntity": [ { "@type": "Question", "name": "Comment évaluer la fonction cellulaire ?", "position": 1, "acceptedAnswer": { "@type": "Answer", "text": "Des tests de culture cellulaire et des analyses biochimiques sont utilisés." } }, { "@type": "Question", "name": "Quels marqueurs indiquent un stress cellulaire ?", "position": 2, "acceptedAnswer": { "@type": "Answer", "text": "Des marqueurs comme la protéine HSP70 et les niveaux de ROS sont souvent mesurés." } }, { "@type": "Question", "name": "Quels tests pour détecter l'apoptose ?", "position": 3, "acceptedAnswer": { "@type": "Answer", "text": "Des tests comme l'Annexin V et la coloration par DAPI sont couramment utilisés." } }, { "@type": "Question", "name": "Comment mesurer la prolifération cellulaire ?", "position": 4, "acceptedAnswer": { "@type": "Answer", "text": "Des méthodes comme le test MTT ou la cytométrie en flux sont efficaces." } }, { "@type": "Question", "name": "Quels signes indiquent une dysfonction mitochondriale ?", "position": 5, "acceptedAnswer": { "@type": "Answer", "text": "Une diminution de l'ATP et une augmentation des espèces réactives de l'oxygène sont des indicateurs." } }, { "@type": "Question", "name": "Quels symptômes d'une inflammation cellulaire ?", "position": 6, "acceptedAnswer": { "@type": "Answer", "text": "Rougeur, chaleur, douleur et gonflement sont des signes d'inflammation." } }, { "@type": "Question", "name": "Comment reconnaître le vieillissement cellulaire ?", "position": 7, "acceptedAnswer": { "@type": "Answer", "text": "Diminution de la régénération et accumulation de dommages ADN sont des indicateurs." } }, { "@type": "Question", "name": "Quels signes d'une nécrose cellulaire ?", "position": 8, "acceptedAnswer": { "@type": "Answer", "text": "Les signes incluent la décoloration, la déformation et la rupture de la membrane cellulaire." } }, { "@type": "Question", "name": "Quels symptômes d'une hypoxie cellulaire ?", "position": 9, "acceptedAnswer": { "@type": "Answer", "text": "Fatigue, essoufflement et cyanose peuvent indiquer une hypoxie au niveau cellulaire." } }, { "@type": "Question", "name": "Quels signes d'une apoptose active ?", "position": 10, "acceptedAnswer": { "@type": "Answer", "text": "La fragmentation de l'ADN et la condensation nucléaire sont des signes d'apoptose." } }, { "@type": "Question", "name": "Comment prévenir le stress cellulaire ?", "position": 11, "acceptedAnswer": { "@type": "Answer", "text": "Une alimentation équilibrée et l'exercice régulier aident à réduire le stress cellulaire." } }, { "@type": "Question", "name": "Quelles habitudes pour éviter le vieillissement cellulaire ?", "position": 12, "acceptedAnswer": { "@type": "Answer", "text": "Éviter le tabac, limiter l'alcool et pratiquer une activité physique régulière sont bénéfiques." } }, { "@type": "Question", "name": "Comment réduire le risque de nécrose cellulaire ?", "position": 13, "acceptedAnswer": { "@type": "Answer", "text": "Maintenir une bonne circulation sanguine et éviter les toxines peut aider." } }, { "@type": "Question", "name": "Quelles stratégies pour prévenir l'hypoxie ?", "position": 14, "acceptedAnswer": { "@type": "Answer", "text": "Éviter les environnements à faible oxygène et pratiquer des exercices respiratoires sont conseillés." } }, { "@type": "Question", "name": "Comment prévenir l'inflammation chronique ?", "position": 15, "acceptedAnswer": { "@type": "Answer", "text": "Une alimentation riche en oméga-3 et des exercices réguliers peuvent réduire l'inflammation." } }, { "@type": "Question", "name": "Comment traiter le stress oxydatif ?", "position": 16, "acceptedAnswer": { "@type": "Answer", "text": "Des antioxydants comme la vitamine C et E peuvent aider à réduire le stress oxydatif." } }, { "@type": "Question", "name": "Quels traitements pour la dysfonction mitochondriale ?", "position": 17, "acceptedAnswer": { "@type": "Answer", "text": "Des cofacteurs comme le coenzyme Q10 et des thérapies ciblées sont souvent utilisés." } }, { "@type": "Question", "name": "Comment gérer l'inflammation cellulaire ?", "position": 18, "acceptedAnswer": { "@type": "Answer", "text": "Des anti-inflammatoires non stéroïdiens (AINS) et des corticostéroïdes sont prescrits." } }, { "@type": "Question", "name": "Quels traitements pour l'apoptose excessive ?", "position": 19, "acceptedAnswer": { "@type": "Answer", "text": "Des inhibiteurs de caspases peuvent être utilisés pour moduler l'apoptose." } }, { "@type": "Question", "name": "Comment traiter les maladies liées au vieillissement cellulaire ?", "position": 20, "acceptedAnswer": { "@type": "Answer", "text": "Des thérapies régénératives et des médicaments ciblant les voies de vieillissement sont explorés." } }, { "@type": "Question", "name": "Quelles complications du stress oxydatif ?", "position": 21, "acceptedAnswer": { "@type": "Answer", "text": "Le stress oxydatif peut entraîner des maladies cardiovasculaires et neurodégénératives." } }, { "@type": "Question", "name": "Quels risques liés à la dysfonction mitochondriale ?", "position": 22, "acceptedAnswer": { "@type": "Answer", "text": "Elle peut causer des troubles métaboliques et des maladies neurodégénératives." } }, { "@type": "Question", "name": "Quelles complications de l'inflammation chronique ?", "position": 23, "acceptedAnswer": { "@type": "Answer", "text": "L'inflammation chronique peut mener à des maladies auto-immunes et des cancers." } }, { "@type": "Question", "name": "Quels effets de l'apoptose excessive ?", "position": 24, "acceptedAnswer": { "@type": "Answer", "text": "Une apoptose excessive peut contribuer à des maladies dégénératives et au cancer." } }, { "@type": "Question", "name": "Quelles complications du vieillissement cellulaire ?", "position": 25, "acceptedAnswer": { "@type": "Answer", "text": "Le vieillissement cellulaire est associé à des maladies comme l'Alzheimer et le diabète." } }, { "@type": "Question", "name": "Quels facteurs de risque pour le stress cellulaire ?", "position": 26, "acceptedAnswer": { "@type": "Answer", "text": "Le tabagisme, l'obésité et le manque d'exercice augmentent le stress cellulaire." } }, { "@type": "Question", "name": "Quels facteurs influencent le vieillissement cellulaire ?", "position": 27, "acceptedAnswer": { "@type": "Answer", "text": "La génétique, l'alimentation et l'exposition aux toxines jouent un rôle clé." } }, { "@type": "Question", "name": "Quels risques liés à l'exposition aux toxines ?", "position": 28, "acceptedAnswer": { "@type": "Answer", "text": "L'exposition à des produits chimiques peut provoquer des dommages cellulaires et des cancers." } }, { "@type": "Question", "name": "Quels facteurs de risque pour l'hypoxie ?", "position": 29, "acceptedAnswer": { "@type": "Answer", "text": "Les maladies respiratoires et les environnements à faible oxygène augmentent le risque d'hypoxie." } }, { "@type": "Question", "name": "Quels facteurs de risque pour l'inflammation ?", "position": 30, "acceptedAnswer": { "@type": "Answer", "text": "Le stress, l'alimentation déséquilibrée et le manque d'exercice sont des facteurs de risque." } } ] } ] }
Dr Olivier Menir

Contenu validé par Dr Olivier Menir

Expert en Médecine, Optimisation des Parcours de Soins et Révision Médicale


Validation scientifique effectuée le 02/02/2025

Contenu vérifié selon les dernières recommandations médicales

Sous-catégories

98 au total
└─

Autophagie

Autophagy D001343 - G04.011
└─

Adhérence cellulaire

Cell Adhesion D002448 - G04.022
└─

Compétition intercellulaire

Cell Competition D000084502 - G04.033
└─

Communication cellulaire

Cell Communication D002450 - G04.085
└─

Compartimentation cellulaire

Cell Compartmentation D002451 - G04.128
└─

Numération cellulaire

Cell Count D002452 - G04.140
└─

Cycle cellulaire

Cell Cycle D002453 - G04.144
└─

Mort cellulaire

Cell Death D016923 - G04.146
└─

Dédifférenciation cellulaire

Cell Dedifferentiation D054337 - G04.148
└─

Différenciation cellulaire

Cell Differentiation D002454 - G04.152
└─

Fusion cellulaire

Cell Fusion D002459 - G04.155
└─

Mécanismes conduisant à la présence d'une cellule dans une autre

Cell-in-Cell Formation D057686 - G04.165
└─

Perméabilité des membranes cellulaires

Cell Membrane Permeability D002463 - G04.175
└─

Polarité de la cellule

Cell Polarity D016764 - G04.250
└─

Respiration cellulaire

Cell Respiration D019069 - G04.270
└─

Forme de la cellule

Cell Shape D048430 - G04.320
└─

Taille de la cellule

Cell Size D048429 - G04.325
└─

Survie cellulaire

Cell Survival D002470 - G04.346
└─

Transdifférenciation cellulaire

Cell Transdifferentiation D054338 - G04.356
└─

Microenvironnement cellulaire

Cellular Microenvironment D060833 - G04.366
└─

Inhibition de contact

Contact Inhibition D003260 - G04.383
└─

Mouvement de cyclose

Cytoplasmic Streaming D003595 - G04.392
└─

Endocytose

Endocytosis D004705 - G04.417
└─

Stress du réticulum endoplasmique

Endoplasmic Reticulum Stress D059865 - G04.434
└─

Exocytose

Exocytosis D005089 - G04.468
└─

Fluidité membranaire

Membrane Fluidity D008560 - G04.570
└─

Fusion membranaire

Membrane Fusion D008561 - G04.575
└─

Gonflement mitochondrial

Mitochondrial Swelling D008933 - G04.590
└─

Renouvellement des mitochondries

Mitochondrial Turnover D063269 - G04.599
└─

Forme de l'organelle

Organelle Shape D053140 - G04.655
└─

Taille de l'organelle

Organelle Size D053141 - G04.670
└─

Agrégation des récepteurs

Receptor Aggregation D011940 - G04.774
└─

Interactions entre récepteurs

Receptor Cross-Talk D020239 - G04.794
└─

Transduction du signal

Signal Transduction D015398 - G04.835
└─└─

Autophagie médiée par les chaperonnes

Chaperone-Mediated Autophagy D000080642 - G04.011.625
└─└─

Macroautophagie

Macroautophagy D000080550 - G04.011.750
└─└─

Microautophagie

Microautophagy D000080551 - G04.011.875
└─└─

Communication autocrine

Autocrine Communication D019898 - G04.085.100
└─└─

Effet bystander

Bystander Effect D024201 - G04.085.155
└─└─

Communication paracrine

Paracrine Communication D019899 - G04.085.600
└─└─

Positionnement des chromosomes

Chromosome Positioning D045584 - G04.128.180
└─└─

Numération des spermatozoïdes

Sperm Count D013076 - G04.140.870
└─└─

Points de contrôle du cycle cellulaire

Cell Cycle Checkpoints D059447 - G04.144.109
└─└─

Interphase

Interphase D007399 - G04.144.500
└─└─

Inversion de la mort cellulaire

Cell Death Reversal D000091442 - G04.146.477
└─└─

Mort cellulaire régulée

Regulated Cell Death D000079404 - G04.146.954
└─└─

Adipogenèse

Adipogenesis D050156 - G04.152.149
└─└─

Empéripolèse

Emperipolesis D057687 - G04.165.500
└─└─

Entose

Entosis D057691 - G04.165.750
└─└─

Agrégation cellulaire

Cell Aggregation D002449 - G04.198.251
└─└─

Inhibition de la migration cellulaire

Cell Migration Inhibition D002464 - G04.198.337
└─└─

Mobilité des spermatozoïdes

Sperm Motility D013081 - G04.198.750
└─└─

Hypoxie cellulaire

Cell Hypoxia D015687 - G04.270.300
└─└─

Stimulation du métabolisme oxydatif

Respiratory Burst D016897 - G04.270.620
└─└─

Effet Warburg en oncologie

Warburg Effect, Oncologic D000082802 - G04.270.810
└─└─

Volume plaquettaire moyen

Mean Platelet Volume D063847 - G04.325.500
└─└─

Plasticité cellulaire

Cell Plasticity D000066670 - G04.356.250
└─└─

Transition épithélio-mésenchymateuse

Epithelial-Mesenchymal Transition D058750 - G04.356.500
└─└─

Niche de cellules souches

Stem Cell Niche D055153 - G04.366.249
└─└─

Microenvironnement tumoral

Tumor Microenvironment D059016 - G04.366.500
└─└─

Pinocytose

Pinocytosis D010873 - G04.417.370
└─└─

Dégranulation cellulaire

Cell Degranulation D015550 - G04.468.160
└─└─

Voie de sécrétion

Secretory Pathway D055571 - G04.468.580
└─└─

Transcytose

Transcytosis D057900 - G04.468.790
└─└─

Dégradation des mitochondries

Mitophagy D063306 - G04.599.500
└─└─

Dynamique mitochondriale

Mitochondrial Dynamics D063154 - G04.599.750
└─└─

Forme du noyau cellulaire

Cell Nucleus Shape D053144 - G04.655.270
└─└─

Taille du noyau cellulaire

Cell Nucleus Size D053145 - G04.670.160
└─└─

Taille de la mitochondrie

Mitochondrial Size D053142 - G04.670.560
└─└─

Voie de signalisation Hippo

Hippo Signaling Pathway D000090683 - G04.835.300
└─└─

Phototransduction

Light Signal Transduction D055537 - G04.835.480
└─└─

Système de signalisation des MAP kinases

MAP Kinase Signaling System D020935 - G04.835.560
└─└─

Mécanotransduction cellulaire

Mechanotransduction, Cellular D040542 - G04.835.580
└─└─

Système cholinergique non neuronal

Non-Neuronal Cholinergic System D000070634 - G04.835.690
└─└─

Systèmes de seconds messagers

Second Messenger Systems D015290 - G04.835.800
└─└─

Voie de signalisation Wnt

Wnt Signaling Pathway D060449 - G04.835.925
└─└─└─

Cytocinèse

Cytokinesis D048749 - G04.144.220.250
└─└─└─

Phase G0

Resting Phase, Cell Cycle D016192 - G04.144.500.300
└─└─└─

Phase G1

G1 Phase D016193 - G04.144.500.320
└─└─└─

Phase G2

G2 Phase D016195 - G04.144.500.340
└─└─└─

Apoptose

Apoptosis D017209 - G04.146.954.035
└─└─└─

Mort cellulaire par autophagie

Autophagic Cell Death D000080549 - G04.146.954.053
└─└─└─

Ferroptose

Ferroptosis D000079403 - G04.146.954.178
└─└─└─

Mort cellulaire immunogène

Immunogenic Cell Death D000079527 - G04.146.954.285
└─└─└─

Nécrose induite par la perméabilité transmembranaire mitochondriale

Mitochondrial Transmembrane Permeability-Driven Necrosis D000079530 - G04.146.954.393
└─└─└─

Nécroptose

Necroptosis D000079302 - G04.146.954.500
└─└─└─

Parthanatos

Parthanatos D000079323 - G04.146.954.750
└─└─└─

Chimiotaxie des leucocytes

Chemotaxis, Leukocyte D002634 - G04.198.424.233
└─└─└─

Hypoxie tumorale

Tumor Hypoxia D000072258 - G04.270.300.500
└─└─└─

Migration transcellulaire des cellules

Transcellular Cell Migration D057688 - G04.468.790.500
└─└─└─

Signalisation calcique

Calcium Signaling D020013 - G04.835.800.100
└─└─└─└─

Points de contrôle de la phase G1 du cycle cellulaire

G1 Phase Cell Cycle Checkpoints D059585 - G04.144.500.320.500
└─└─└─└─

Points de contrôle de la phase G2 du cycle cellulaire

G2 Phase Cell Cycle Checkpoints D059565 - G04.144.500.340.500
└─└─└─└─

Points de contrôle de la phase S du cycle cellulaire

S Phase Cell Cycle Checkpoints D059807 - G04.144.500.800.500
└─└─└─└─

Anoïkis

Anoikis D023102 - G04.146.954.035.060
└─└─└─└─

Éryptose

Eryptosis D000072817 - G04.146.954.035.295
└─└─└─└─

Pyroptose

Pyroptosis D000069292 - G04.146.954.035.530
└─└─└─└─

Roulement des leucocytes

Leukocyte Rolling D036904 - G04.198.424.233.500

Auteurs principaux

Stefania E Kapsetaki

2 publications dans cette catégorie

Affiliations :
  • Arizona Cancer Evolution Center, Arizona State University, Tempe, AZ, USA. stefania.kapsetaki@tufts.edu.
  • Biodesign Center for Biocomputing, Security and Society, Arizona State University, Tempe, AZ, USA. stefania.kapsetaki@tufts.edu.
  • Department of Biology, School of Arts and Sciences, Tufts University, Medford, MA, USA. stefania.kapsetaki@tufts.edu.
Publications dans "Phénomènes physiologiques cellulaires" :

Luis H Cisneros

2 publications dans cette catégorie

Affiliations :
  • Arizona Cancer Evolution Center, Arizona State University, Tempe, AZ, USA.
  • Biodesign Center for Biocomputing, Security and Society, Arizona State University, Tempe, AZ, USA.
  • School of Life Sciences, Arizona State University, Tempe, AZ, USA.
Publications dans "Phénomènes physiologiques cellulaires" :

Carlo C Maley

2 publications dans cette catégorie

Affiliations :
  • Arizona Cancer Evolution Center, Arizona State University, Tempe, AZ, USA.
  • Biodesign Center for Biocomputing, Security and Society, Arizona State University, Tempe, AZ, USA.
  • School of Life Sciences, Arizona State University, Tempe, AZ, USA.
  • Biodesign Center for Mechanisms of Evolution, Arizona State University, Tempe, AZ, USA.
  • Center for Evolution and Medicine, Arizona State University, Tempe, AZ, USA.
Publications dans "Phénomènes physiologiques cellulaires" :

Oliver McNeilly

1 publication dans cette catégorie

Affiliations :
  • Australian Institute of Microbiology and Infection, University of Technology Sydney, Broadway, New South Wales, Australia.
Publications dans "Phénomènes physiologiques cellulaires" :

Riti Mann

1 publication dans cette catégorie

Affiliations :
  • Australian Institute of Microbiology and Infection, University of Technology Sydney, Broadway, New South Wales, Australia.
Publications dans "Phénomènes physiologiques cellulaires" :

Max Laurence Cummins

1 publication dans cette catégorie

Affiliations :
  • Australian Institute of Microbiology and Infection, University of Technology Sydney, Broadway, New South Wales, Australia.
  • Australian Centre for Genomic Epidemiological Microbiology, University of Technology Sydney, Broadway, New South Wales, Australia.
Publications dans "Phénomènes physiologiques cellulaires" :

Steven P Djordjevic

1 publication dans cette catégorie

Affiliations :
  • Australian Institute of Microbiology and Infection, University of Technology Sydney, Broadway, New South Wales, Australia.
  • Australian Centre for Genomic Epidemiological Microbiology, University of Technology Sydney, Broadway, New South Wales, Australia.
Publications dans "Phénomènes physiologiques cellulaires" :

Mehrad Hamidian

1 publication dans cette catégorie

Affiliations :
  • Australian Institute of Microbiology and Infection, University of Technology Sydney, Broadway, New South Wales, Australia.
Publications dans "Phénomènes physiologiques cellulaires" :

Cindy Gunawan

1 publication dans cette catégorie

Affiliations :
  • Australian Institute of Microbiology and Infection, University of Technology Sydney, Broadway, New South Wales, Australia.
  • School of Chemical Engineering, University of New South Wales, Sydney, New South Wales, Australia.
Publications dans "Phénomènes physiologiques cellulaires" :

Mesalie Feleke

1 publication dans cette catégorie

Affiliations :
  • Division of Regenerative Biology, School of Biomedical Sciences, University of Western Australia, Perth, 6009, Australia.
Publications dans "Phénomènes physiologiques cellulaires" :

Samuel Bennett

1 publication dans cette catégorie

Affiliations :
  • Division of Regenerative Biology, School of Biomedical Sciences, University of Western Australia, Perth, 6009, Australia.
Publications dans "Phénomènes physiologiques cellulaires" :

Jiazhi Chen

1 publication dans cette catégorie

Affiliations :
  • Guangdong Provincial Key Laboratory of Industrial Surfactant, Guangdong Research Institute of Petrochemical and Fine Chemical Engineering, Guangdong Academy of Sciences, Guangzhou, 510665, China.
  • Division of Regenerative Biology, School of Biomedical Sciences, University of Western Australia, Perth, 6009, Australia.
Publications dans "Phénomènes physiologiques cellulaires" :

Xiaoyong Hu

1 publication dans cette catégorie

Affiliations :
  • Guangdong Provincial Key Laboratory of Industrial Surfactant, Guangdong Research Institute of Petrochemical and Fine Chemical Engineering, Guangdong Academy of Sciences, Guangzhou, 510665, China.
Publications dans "Phénomènes physiologiques cellulaires" :

Desmond Williams

1 publication dans cette catégorie

Affiliations :
  • Division of Regenerative Biology, School of Biomedical Sciences, University of Western Australia, Perth, 6009, Australia.
Publications dans "Phénomènes physiologiques cellulaires" :

Jiake Xu

1 publication dans cette catégorie

Affiliations :
  • Division of Regenerative Biology, School of Biomedical Sciences, University of Western Australia, Perth, 6009, Australia.
Publications dans "Phénomènes physiologiques cellulaires" :

Innokentii E Vishnyakov

1 publication dans cette catégorie

Affiliations :
  • Institute of Cytology, Russian Academy of Sciences, 194064 St. Petersburg, Russia.
Publications dans "Phénomènes physiologiques cellulaires" :

Karol Borensztejn

1 publication dans cette catégorie

Affiliations :
  • Histology and Embryology Students' Science Association, Department of Histology and Embryology, Faculty of Medicine, Warsaw Medical University, Chalubinskiego 5, 02-004 Warsaw, Poland.
Publications dans "Phénomènes physiologiques cellulaires" :

Paweł Tyrna

1 publication dans cette catégorie

Affiliations :
  • Histology and Embryology Students' Science Association, Department of Histology and Embryology, Faculty of Medicine, Warsaw Medical University, Chalubinskiego 5, 02-004 Warsaw, Poland.
Publications dans "Phénomènes physiologiques cellulaires" :

Agata M Gaweł

1 publication dans cette catégorie

Affiliations :
  • Histology and Embryology Students' Science Association, Department of Histology and Embryology, Faculty of Medicine, Warsaw Medical University, Chalubinskiego 5, 02-004 Warsaw, Poland.
Publications dans "Phénomènes physiologiques cellulaires" :

Ireneusz Dziuba

1 publication dans cette catégorie

Affiliations :
  • Faculty of Medicine, Collegium Medicum, Cardinal Stefan Wyszyński University in Warsaw, Dewajtis 5, 01-815 Warsaw, Poland.
  • Faculty of Medicine, University of Technology, Rolna 43, 40-555 Katowice, Poland.
Publications dans "Phénomènes physiologiques cellulaires" :

Sources (10000 au total)