Screening persistent organic pollutants for effects on testosterone and estrogen synthesis at human-relevant concentrations using H295R cells in 96-well plates.


Journal

Cell biology and toxicology
ISSN: 1573-6822
Titre abrégé: Cell Biol Toxicol
Pays: Switzerland
ID NLM: 8506639

Informations de publication

Date de publication:
13 Aug 2024
Historique:
received: 12 03 2024
accepted: 18 07 2024
medline: 13 8 2024
pubmed: 13 8 2024
entrez: 13 8 2024
Statut: epublish

Résumé

Many persistent organic pollutants (POPs) are suspected endocrine disruptors and it is important to investigate their effects at low concentrations relevant to human exposure. Here, the OECD test guideline #456 steroidogenesis assay was downscaled to a 96-well microplate format to screen 24 POPs for their effects on viability, and testosterone and estradiol synthesis using the human adrenocortical cell line H295R. The compounds (six polyfluoroalkyl substances, five organochlorine pesticides, ten polychlorinated biphenyls and three polybrominated diphenyl ethers) were tested at human-relevant levels (1 nM to 10 µM). Increased estradiol synthesis, above the OECD guideline threshold of 1.5-fold solvent control, was shown after exposure to 10 µM PCB-156 (153%) and PCB-180 (196%). Interestingly, the base hormone synthesis varied depending on the cell batch. An alternative data analysis using a linear mixed-effects model that include multiple independent experiments and considers batch-dependent variation was therefore applied. This approach revealed small but statistically significant effects on estradiol or testosterone synthesis for 17 compounds. Increased testosterone levels were demonstrated even at 1 nM for PCB-74 (18%), PCB-99 (29%), PCB-118 (16%), PCB-138 (19%), PCB-180 (22%), and PBDE-153 (21%). The MTT assay revealed significant effects on cell viability after exposure to 1 nM of perfluoroundecanoic acid (12%), 3 nM PBDE-153 (9%), and 10 µM of PCB-156 (6%). This shows that some POPs can interfere with endocrine signaling at concentrations found in human blood, highlighting the need for further investigation into the toxicological mechanisms of POPs and their mixtures at low concentrations relevant to human exposure.

Identifiants

pubmed: 39136868
doi: 10.1007/s10565-024-09902-4
pii: 10.1007/s10565-024-09902-4
doi:

Substances chimiques

Testosterone 3XMK78S47O
Persistent Organic Pollutants 0
Endocrine Disruptors 0
Polychlorinated Biphenyls DFC2HB4I0K
Halogenated Diphenyl Ethers 0
Estradiol 4TI98Z838E
Estrogens 0
Pesticides 0
Hydrocarbons, Chlorinated 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

69

Subventions

Organisme : Svenska Forskningsrådet Formas
ID : 2018-02268
Organisme : Svenska Forskningsrådet Formas
ID : 2018-02268
Organisme : Svenska Forskningsrådet Formas
ID : 2018-02268
Organisme : Svenska Forskningsrådet Formas
ID : 2018-02268
Organisme : Svenska Forskningsrådet Formas
ID : 2018-02268

Informations de copyright

© 2024. The Author(s).

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Auteurs

Denise Strand (D)

Science for Life Laboratory, Department of Environmental Science, Stockholm University, 114 18, Stockholm, Sweden.

Erik Nylander (E)

Science for Life Laboratory, Department of Environmental Science, Stockholm University, 114 18, Stockholm, Sweden.

Andrey Höglund (A)

Science for Life Laboratory, Department of Environmental Science, Stockholm University, 114 18, Stockholm, Sweden.

Bo Lundgren (B)

Science for Life Laboratory, Biochemical and Cellular Assay unit, Dept. of Biochemistry and Biophysics, Stockholm University, 106 91, Stockholm, Sweden.

Jonathan W Martin (JW)

Science for Life Laboratory, Department of Environmental Science, Stockholm University, 114 18, Stockholm, Sweden.

Oskar Karlsson (O)

Science for Life Laboratory, Department of Environmental Science, Stockholm University, 114 18, Stockholm, Sweden. Oskar.Karlsson@aces.su.se.

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