Endothelial Cell Phenotypes Demonstrate Different Metabolic Patterns and Predict Mortality in Trauma Patients.

endotheliopathy genome-scale metabolic model metabolomics systems biology trauma tricarboxylic acid cycle

Journal

International journal of molecular sciences
ISSN: 1422-0067
Titre abrégé: Int J Mol Sci
Pays: Switzerland
ID NLM: 101092791

Informations de publication

Date de publication:
23 Jan 2023
Historique:
received: 09 11 2022
revised: 15 01 2023
accepted: 19 01 2023
entrez: 11 2 2023
pubmed: 12 2 2023
medline: 15 2 2023
Statut: epublish

Résumé

In trauma patients, shock-induced endotheliopathy (SHINE) is associated with a poor prognosis. We have previously identified four metabolic phenotypes in a small cohort of trauma patients (N = 20) and displayed the intracellular metabolic profile of the endothelial cell by integrating quantified plasma metabolomic profiles into a genome-scale metabolic model (iEC-GEM). A retrospective observational study of 99 trauma patients admitted to a Level 1 Trauma Center. Mass spectrometry was conducted on admission samples of plasma metabolites. Quantified metabolites were analyzed by computational network analysis of the iEC-GEM. Four plasma metabolic phenotypes (A-D) were identified, of which phenotype D was associated with an increased injury severity score (

Identifiants

pubmed: 36768579
pii: ijms24032257
doi: 10.3390/ijms24032257
pmc: PMC9916682
pii:
doi:

Types de publication

Observational Study Journal Article

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : NIGMS NIH HHS
ID : T32 GM008792
Pays : United States

Déclaration de conflit d'intérêts

The authors declare no conflict of interest.

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Auteurs

Hanne H Henriksen (HH)

Section for Transfusion Medicine, Capital Region Blood Bank, Rigshospitalet, University of Copenhagen, 2100 Copenhagen, Denmark.
CAG Center for Endotheliomics, Copenhagen University Hospital, Rigshospitalet, 2100 Copenhagen, Denmark.

Igor Marín de Mas (I)

Novo Nordisk Foundation Center for Biosustainability, Technical University of Denmark, 2800 Kongens Lyngby, Denmark.

Lars K Nielsen (LK)

Novo Nordisk Foundation Center for Biosustainability, Technical University of Denmark, 2800 Kongens Lyngby, Denmark.
Australian Institute for Bioengineering and Nanotechnology (AIBN), The University of Queensland, 4072 Brisbane, Australia.

Joseph Krocker (J)

Center for Translational Injury Research, Department of Surgery, University of Texas Health Science Center, Houston, TX 77030, USA.

Jakob Stensballe (J)

Section for Transfusion Medicine, Capital Region Blood Bank, Rigshospitalet, University of Copenhagen, 2100 Copenhagen, Denmark.
CAG Center for Endotheliomics, Copenhagen University Hospital, Rigshospitalet, 2100 Copenhagen, Denmark.
Department of Anesthesia and Trauma Center, Center of Head and Orthopedics, Rigshospitalet, 2100 Copenhagen, Denmark.

Sigurður T Karvelsson (ST)

Center for Systems Biology, University of Iceland, 101 Reykjavik, Iceland.

Niels H Secher (NH)

Department of Anesthesiology, Centre for Cancer and Organ Diseases, Rigshospitalet, University of Copenhagen, 2100 Copenhagen, Denmark.

Óttar Rolfsson (Ó)

Center for Systems Biology, University of Iceland, 101 Reykjavik, Iceland.

Charles E Wade (CE)

Center for Translational Injury Research, Department of Surgery, University of Texas Health Science Center, Houston, TX 77030, USA.

Pär I Johansson (PI)

Section for Transfusion Medicine, Capital Region Blood Bank, Rigshospitalet, University of Copenhagen, 2100 Copenhagen, Denmark.
CAG Center for Endotheliomics, Copenhagen University Hospital, Rigshospitalet, 2100 Copenhagen, Denmark.
Center for Translational Injury Research, Department of Surgery, University of Texas Health Science Center, Houston, TX 77030, USA.
Center for Systems Biology, University of Iceland, 101 Reykjavik, Iceland.

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