BLMP-1 promotes developmental cell death in C. elegans by timely repression of ced-9 transcription.


Journal

Development (Cambridge, England)
ISSN: 1477-9129
Titre abrégé: Development
Pays: England
ID NLM: 8701744

Informations de publication

Date de publication:
15 10 2021
Historique:
received: 17 06 2020
accepted: 14 09 2021
pubmed: 21 9 2021
medline: 15 12 2021
entrez: 20 9 2021
Statut: ppublish

Résumé

Programmed cell death (PCD) is a common cell fate in metazoan development. PCD effectors are extensively studied, but how they are temporally regulated is less understood. Here, we report a mechanism controlling tail-spike cell death onset during Caenorhabditis elegans development. We show that the zinc-finger transcription factor BLMP-1, which controls larval development timing, also regulates embryonic tail-spike cell death initiation. BLMP-1 functions upstream of CED-9 and in parallel to DRE-1, another CED-9 and tail-spike cell death regulator. BLMP-1 expression is detected in the tail-spike cell shortly after the cell is born, and blmp-1 mutations promote ced-9-dependent tail-spike cell survival. BLMP-1 binds ced-9 gene regulatory sequences, and inhibits ced-9 transcription just before cell-death onset. BLMP-1 and DRE-1 function together to regulate developmental timing, and their mammalian homologs regulate B-lymphocyte fate. Our results, therefore, identify roles for developmental timing genes in cell-death initiation, and suggest conservation of these functions.

Identifiants

pubmed: 34541605
pii: 272221
doi: 10.1242/dev.193995
pmc: PMC8572009
pii:
doi:

Substances chimiques

Caenorhabditis elegans Proteins 0
Repressor Proteins 0
blmp-1 protein, C elegans 0

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : NICHD NIH HHS
ID : F32 HD089640
Pays : United States
Organisme : NINDS NIH HHS
ID : R35 NS105094
Pays : United States

Informations de copyright

© 2021. Published by The Company of Biologists Ltd.

Déclaration de conflit d'intérêts

Competing interests The authors declare no competing or financial interests.

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Auteurs

Hang-Shiang Jiang (HS)

Institute of Molecular and Cellular Biology, National Taiwan University, Taipei, 106216, Taiwan.

Piya Ghose (P)

Laboratory of Developmental Genetics, The Rockefeller University, New York, NY 10065, USA.
Department of Biology, The University of Texas at Arlington, Arlington, TX 76019, USA.

Hsiao-Fen Han (HF)

Institute of Molecular and Cellular Biology, National Taiwan University, Taipei, 106216, Taiwan.

Yun-Zhe Wu (YZ)

Institute of Molecular and Cellular Biology, National Taiwan University, Taipei, 106216, Taiwan.

Ya-Yin Tsai (YY)

Institute of Molecular and Cellular Biology, National Taiwan University, Taipei, 106216, Taiwan.

Huang-Chin Lin (HC)

Institute of Molecular and Cellular Biology, National Taiwan University, Taipei, 106216, Taiwan.

Wei-Chin Tseng (WC)

Institute of Molecular and Cellular Biology, National Taiwan University, Taipei, 106216, Taiwan.

Jui-Ching Wu (JC)

Department of Clinical Laboratory Sciences and Medical Biotechnology, College of Medicine, National Taiwan University, Taipei, 100229, Taiwan.

Shai Shaham (S)

Laboratory of Developmental Genetics, The Rockefeller University, New York, NY 10065, USA.

Yi-Chun Wu (YC)

Institute of Molecular and Cellular Biology, National Taiwan University, Taipei, 106216, Taiwan.
Department of Life Science, Center for Systems Biology, and Research Center for Developmental Biology and Regenerative Medicine, National Taiwan University, Taipei, 106216, Taiwan.
Institute of Atomic and Molecular Sciences, Academia Sinica, Taipei, 106216, Taiwan.

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