Diagnostic utility of programmed cell death ligand 1 (clone SP142) immunohistochemistry for malignant lymphoma and lymphoproliferative disorders: A brief review.


Journal

Journal of clinical and experimental hematopathology : JCEH
ISSN: 1880-9952
Titre abrégé: J Clin Exp Hematop
Pays: Japan
ID NLM: 101141257

Informations de publication

Date de publication:
22 Dec 2021
Historique:
pubmed: 14 9 2021
medline: 14 1 2022
entrez: 13 9 2021
Statut: ppublish

Résumé

The programmed cell death 1 (PD1)/PD1 ligand (PD-L1) axis plays an important role in tumor cell escape from immune control and has been most extensively investigated for therapeutic purposes. However, PD-L1 immunohistochemistry is still not used widely for diagnosis. We review the diagnostic utility of PD-L1 (by clone SP142) immunohistochemistry in large-cell lymphomas, mainly consisting of classic Hodgkin lymphoma (CHL) and diffuse large B-cell lymphoma (DLBCL). Neoplastic PD-L1 (nPD-L1) expression on Hodgkin and Reed-Sternberg cells is well-established among prototypic CHL. Of note, EBV+ CHL often poses a challenge for differential diagnosis from peripheral T-cell lymphoma with EBV+ non-malignant large B-cells; their distinction is based on the lack of PD-L1 expression on large B-cells in the latter. The nPD-L1 expression further provides a good diagnostic consensus for CHL with primary extranodal disease conceivably characterized by a combined pathogenesis of immune escape of tumor cells and immunodeficiency. Compared with CHL, the nPD-L1 expression rate is much lower in DLBCL, highlighting some specific subgroups of intravascular large B-cell lymphoma, primary mediastinal large B-cell lymphoma, and EBV+ DLBCL. They consist of nPD-L1-positive and -negative subgroups, but their clinicopathological significance remains to be elucidated. Microenvironmental PD-L1 positivity on immune cells may be associated with a favorable prognosis in extranodal DLBCL. PD-L1 (by SP142) immunohistochemistry has helped us to understand the immune biology of lymphoid neoplasms possibly related by immune escape and/or immunodeficiency. However, knowledge of these issues remains limited and should be clarified for diagnostic consensus in the future.

Identifiants

pubmed: 34511582
doi: 10.3960/jslrt.21003
pmc: PMC8808108
doi:

Substances chimiques

B7-H1 Antigen 0
Biomarkers, Tumor 0
Ligands 0

Types de publication

Journal Article Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

182-191

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Auteurs

Ayako Sakakibara (A)

Department of Pathology and Laboratory Medicine, Nagoya University Hospital, Nagoya, Japan.

Kei Kohno (K)

Department of Pathology and Laboratory Medicine, Nagoya University Hospital, Nagoya, Japan.

Eri Ishikawa (E)

Department of Pathology and Laboratory Medicine, Nagoya University Hospital, Nagoya, Japan.

Yuka Suzuki (Y)

Department of Pathology and Laboratory Medicine, Nagoya University Hospital, Nagoya, Japan.

Yuta Tsuyuki (Y)

Department of Pathology and Laboratory Medicine, Nagoya University Hospital, Nagoya, Japan.

Satoko Shimada (S)

Department of Pathology and Laboratory Medicine, Nagoya University Hospital, Nagoya, Japan.

Kazuyuki Shimada (K)

Department of Hematology and Oncology, Nagoya University Graduate School of Medicine, Nagoya, Japan.

Akira Satou (A)

Department of Surgical Pathology, Aichi Medical University Hospital, Nagakute, Japan.

Taishi Takahara (T)

Department of Surgical Pathology, Aichi Medical University Hospital, Nagakute, Japan.

Akiko Ohashi (A)

Department of Surgical Pathology, Aichi Medical University Hospital, Nagakute, Japan.

Emiko Takahashi (E)

Department of Surgical Pathology, Aichi Medical University Hospital, Nagakute, Japan.

Seiichi Kato (S)

Department of Pathology and Molecular Diagnostics, Aichi Cancer Center Hospital.

Shigeo Nakamura (S)

Department of Pathology and Laboratory Medicine, Nagoya University Hospital, Nagoya, Japan.

Naoko Asano (N)

Department of Clinical Laboratory, Nagano Prefectural Suzaka Hospital, Nagano, Japan.

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