Recurrent membranous nephropathy after transplantation: donor antigen and HLA converge in defining risk.


Journal

Kidney international
ISSN: 1523-1755
Titre abrégé: Kidney Int
Pays: United States
ID NLM: 0323470

Informations de publication

Date de publication:
03 2021
Historique:
received: 18 10 2020
accepted: 29 10 2020
entrez: 27 2 2021
pubmed: 28 2 2021
medline: 6 3 2021
Statut: ppublish

Résumé

Membranous nephropathy, like many forms of glomerulonephritis, is an HLA-associated autoimmune disease that can recur in the transplanted kidney. In this issue of Kidney International, Berchtold and colleagues publish an intriguing and important paper on risk factors for recurrent post-transplant membranous nephropathy due to autoimmunity to PLA2R1. They found that the genetics of both the autoantigen and donor HLA are important determinants of the risk of recurrent disease in the graft.

Identifiants

pubmed: 33637201
pii: S0085-2538(20)31415-0
doi: 10.1016/j.kint.2020.10.044
pii:
doi:

Substances chimiques

PLA2R1 protein, human 0
Receptors, Phospholipase A2 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't Comment

Langues

eng

Sous-ensembles de citation

IM

Pagination

545-548

Commentaires et corrections

Type : CommentOn

Informations de copyright

Copyright © 2020 International Society of Nephrology. Published by Elsevier Inc. All rights reserved.

Auteurs

Kate J Robson (KJ)

Centre for Inflammatory Diseases, Monash University Department of Medicine, Monash Medical Centre, Monash University, Clayton, Victoria, Australia; Department of Nephrology, Monash Health, Clayton, Victoria, Australia.

A Richard Kitching (AR)

Centre for Inflammatory Diseases, Monash University Department of Medicine, Monash Medical Centre, Monash University, Clayton, Victoria, Australia; Department of Nephrology, Monash Health, Clayton, Victoria, Australia; Department of Pediatric Nephrology, Monash Health, Clayton, Victoria, Australia. Electronic address: richard.kitching@monash.edu.

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Classifications MeSH