Classification of node-positive melanomas into prognostic subgroups using keratin, immune, and melanogenesis expression patterns.


Journal

Oncogene
ISSN: 1476-5594
Titre abrégé: Oncogene
Pays: England
ID NLM: 8711562

Informations de publication

Date de publication:
03 2021
Historique:
received: 10 06 2020
accepted: 18 01 2021
revised: 08 11 2020
pubmed: 11 2 2021
medline: 16 10 2021
entrez: 10 2 2021
Statut: ppublish

Résumé

Cutaneous melanoma tumors are heterogeneous and show diverse responses to treatment. Identification of robust molecular biomarkers for classifying melanoma tumors into clinically distinct and homogenous subtypes is crucial for improving the diagnosis and treatment of the disease. In this study, we present a classification of melanoma tumors into four subtypes with different survival profiles based on three distinct gene expression signatures: keratin, immune, and melanogenesis. The melanogenesis expression pattern includes several genes that are characteristic of the melanosome organelle and correlates with worse survival, suggesting the involvement of melanosomes in melanoma aggression. We experimentally validated the secretion of melanosomes into surrounding tissues by melanoma tumors, which potentially affects the lethality of metastasis. We propose a simple molecular decision tree classifier for predicting a tumor's subtype based on representative genes from the three identified signatures. Key predictor genes were experimentally validated on melanoma samples taken from patients with varying survival outcomes. Our three-pattern approach for classifying melanoma tumors can contribute to advancing the understanding of melanoma variability and promote accurate diagnosis, prognostication, and treatment.

Identifiants

pubmed: 33564068
doi: 10.1038/s41388-021-01665-0
pii: 10.1038/s41388-021-01665-0
pmc: PMC7946641
doi:

Substances chimiques

Melanins 0
Muscle Proteins 0
Neoplasm Proteins 0
Receptors, Immunologic 0
TIGIT protein, human 0
Tripartite Motif Proteins 0
TRIM63 protein, human EC 2.3.2.27
Ubiquitin-Protein Ligases EC 2.3.2.27
KLK8 protein, human EC 3.4.21.-
Kallikreins EC 3.4.21.-

Types de publication

Journal Article Research Support, Non-U.S. Gov't Research Support, U.S. Gov't, Non-P.H.S.

Langues

eng

Sous-ensembles de citation

IM

Pagination

1792-1805

Subventions

Organisme : European Research Council
ID : 726225
Pays : International

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Auteurs

Dvir Netanely (D)

Blavatnik School of Computer Science, Tel Aviv University, Tel Aviv, Israel.

Stav Leibou (S)

Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel.

Roma Parikh (R)

Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel.

Neta Stern (N)

Blavatnik School of Computer Science, Tel Aviv University, Tel Aviv, Israel.

Hananya Vaknine (H)

Department of Oncology, Edith Wolfson Medical Center, Holon, Israel.

Ronen Brenner (R)

Department of Oncology, Edith Wolfson Medical Center, Holon, Israel.

Sarah Amar (S)

Department of Oncology, Edith Wolfson Medical Center, Holon, Israel.

Rivi Haiat Factor (RH)

Department of Plastic and Reconstructive Surgery, Edith Wolfson Medical Center, Holon, Israel.

Tomer Perluk (T)

Department of Plastic and Reconstructive Surgery, Edith Wolfson Medical Center, Holon, Israel.

Jacob Frand (J)

Department of Plastic and Reconstructive Surgery, Edith Wolfson Medical Center, Holon, Israel.

Eran Nizri (E)

Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel.
Department of Surgery A, Tel Aviv Sourasky Medical Center, Tel Aviv, Israel.

Dov Hershkovitz (D)

Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel.
Institute of Pathology, Tel Aviv Sourasky Medical Center, Tel Aviv, Israel.

Valentina Zemser-Werner (V)

Institute of Pathology, Tel Aviv Sourasky Medical Center, Tel Aviv, Israel.

Carmit Levy (C)

Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel.

Ron Shamir (R)

Blavatnik School of Computer Science, Tel Aviv University, Tel Aviv, Israel. rshamir@tau.ac.il.

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Classifications MeSH