Leptin modulates pancreatic β-cell membrane potential through Src kinase-mediated phosphorylation of NMDA receptors.
Animals
Cell Line
Diabetes Mellitus, Experimental
/ genetics
Diabetes Mellitus, Type 2
/ genetics
Humans
Insulin-Secreting Cells
/ metabolism
Leptin
/ genetics
Membrane Potentials
Mice
Mice, Mutant Strains
Mutation
Obesity
/ genetics
Phosphorylation
Receptors, N-Methyl-D-Aspartate
/ genetics
src-Family Kinases
/ genetics
GluN2A
KATP channel
Src
Src kinase
beta cell (B-cell)
glutamate receptor
phosphorylation
type 2 diabetes
β-cells
Journal
The Journal of biological chemistry
ISSN: 1083-351X
Titre abrégé: J Biol Chem
Pays: United States
ID NLM: 2985121R
Informations de publication
Date de publication:
11 12 2020
11 12 2020
Historique:
received:
03
08
2020
revised:
08
10
2020
pubmed:
11
10
2020
medline:
17
3
2021
entrez:
10
10
2020
Statut:
ppublish
Résumé
The adipocyte-derived hormone leptin increases trafficking of K
Identifiants
pubmed: 33037073
pii: S0021-9258(17)50616-X
doi: 10.1074/jbc.RA120.015489
pmc: PMC7863909
pii:
doi:
Substances chimiques
LEP protein, human
0
Leptin
0
NR2B NMDA receptor
0
Receptors, N-Methyl-D-Aspartate
0
src-Family Kinases
EC 2.7.10.2
N-methyl D-aspartate receptor subtype 2A
VH92ICR8HX
Types de publication
Journal Article
Research Support, N.I.H., Extramural
Langues
eng
Sous-ensembles de citation
IM
Pagination
17281-17297Subventions
Organisme : NIH HHS
ID : P51 OD011092
Pays : United States
Organisme : NIDDK NIH HHS
ID : R01 DK057699
Pays : United States
Informations de copyright
© 2020 Cochrane et al.
Déclaration de conflit d'intérêts
Conflict of interest—The authors declare that they have no conflicts of interest with the contents of this article.
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