Metaplastic breast cancers frequently express immune checkpoint markers FOXP3 and PD-L1.


Journal

British journal of cancer
ISSN: 1532-1827
Titre abrégé: Br J Cancer
Pays: England
ID NLM: 0370635

Informations de publication

Date de publication:
11 2020
Historique:
received: 16 04 2020
accepted: 27 08 2020
revised: 19 08 2020
pubmed: 18 9 2020
medline: 19 3 2021
entrez: 17 9 2020
Statut: ppublish

Résumé

Metaplastic breast carcinoma encompasses a heterogeneous group of tumours with differentiation into squamous and/or spindle, chondroid, osseous or rhabdoid mesenchymal-looking elements. Emerging immunotherapies targeting Programmed Death Ligand 1 (PD-L1) and immune-suppressing T cells (Tregs) may benefit metaplastic breast cancer patients, which are typically chemo-resistant and do not express hormone therapy targets. We evaluated the immunohistochemical expression of PD-L1 and FOXP3, and the extent of tumour infiltrating lymphocytes (TILs) in a large cohort of metaplastic breast cancers, with survival data. Metaplastic breast cancers were significantly enriched for PD-L1 positive tumour cells, compared to triple-negative ductal breast cancers (P < 0.0001), while there was no significant difference in PD-L1 positive TILs. Metaplastic breast cancers were also significantly enriched for TILs expressing FOXP3, with FOXP3 positive intra-tumoural TILs (iTILs) associated with an adverse prognostic outcome (P = 0.0226). Multivariate analysis identified FOXP3 iTILs expression status as an important independent prognostic factor for patient survival. Our findings indicate the clinical significance and prognostic value of FOXP3, PD-1/PD-L1 checkpoint and TILs in metaplastic breast cancer and confirm that a subset of metaplastics may benefit from immune-based therapies.

Sections du résumé

BACKGROUND
Metaplastic breast carcinoma encompasses a heterogeneous group of tumours with differentiation into squamous and/or spindle, chondroid, osseous or rhabdoid mesenchymal-looking elements. Emerging immunotherapies targeting Programmed Death Ligand 1 (PD-L1) and immune-suppressing T cells (Tregs) may benefit metaplastic breast cancer patients, which are typically chemo-resistant and do not express hormone therapy targets.
METHODS
We evaluated the immunohistochemical expression of PD-L1 and FOXP3, and the extent of tumour infiltrating lymphocytes (TILs) in a large cohort of metaplastic breast cancers, with survival data.
RESULTS
Metaplastic breast cancers were significantly enriched for PD-L1 positive tumour cells, compared to triple-negative ductal breast cancers (P < 0.0001), while there was no significant difference in PD-L1 positive TILs. Metaplastic breast cancers were also significantly enriched for TILs expressing FOXP3, with FOXP3 positive intra-tumoural TILs (iTILs) associated with an adverse prognostic outcome (P = 0.0226). Multivariate analysis identified FOXP3 iTILs expression status as an important independent prognostic factor for patient survival.
CONCLUSIONS
Our findings indicate the clinical significance and prognostic value of FOXP3, PD-1/PD-L1 checkpoint and TILs in metaplastic breast cancer and confirm that a subset of metaplastics may benefit from immune-based therapies.

Identifiants

pubmed: 32939056
doi: 10.1038/s41416-020-01065-3
pii: 10.1038/s41416-020-01065-3
pmc: PMC7686342
doi:

Substances chimiques

B7-H1 Antigen 0
Biomarkers, Tumor 0
CD274 protein, human 0
FOXP3 protein, human 0
Forkhead Transcription Factors 0
Immune Checkpoint Inhibitors 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1665-1672

Subventions

Organisme : Department of Health | National Health and Medical Research Council (NHMRC)
ID : APP1113867

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Auteurs

Emarene Kalaw (E)

Faculty of Medicine, UQ Centre for Clinical Research, The University of Queensland, Herston, Brisbane, QLD, Australia.

Malcolm Lim (M)

Faculty of Medicine, UQ Centre for Clinical Research, The University of Queensland, Herston, Brisbane, QLD, Australia.
QIMR Berghofer Medical Research Institute, Herston, Brisbane, QLD, Australia.

Jamie R Kutasovic (JR)

Faculty of Medicine, UQ Centre for Clinical Research, The University of Queensland, Herston, Brisbane, QLD, Australia.
QIMR Berghofer Medical Research Institute, Herston, Brisbane, QLD, Australia.

Anna Sokolova (A)

Faculty of Medicine, UQ Centre for Clinical Research, The University of Queensland, Herston, Brisbane, QLD, Australia.
Pathology Queensland, The Royal Brisbane & Women's Hospital, Herston, Brisbane, QLD, Australia.

Lucinda Taege (L)

Faculty of Medicine, UQ Centre for Clinical Research, The University of Queensland, Herston, Brisbane, QLD, Australia.
Pathology Queensland, The Royal Brisbane & Women's Hospital, Herston, Brisbane, QLD, Australia.

Kate Johnstone (K)

Faculty of Medicine, UQ Centre for Clinical Research, The University of Queensland, Herston, Brisbane, QLD, Australia.
Pathology Queensland, The Royal Brisbane & Women's Hospital, Herston, Brisbane, QLD, Australia.

James Bennett (J)

Faculty of Medicine, UQ Centre for Clinical Research, The University of Queensland, Herston, Brisbane, QLD, Australia.
Pathology Queensland, The Royal Brisbane & Women's Hospital, Herston, Brisbane, QLD, Australia.

Jodi M Saunus (JM)

Faculty of Medicine, UQ Centre for Clinical Research, The University of Queensland, Herston, Brisbane, QLD, Australia.
QIMR Berghofer Medical Research Institute, Herston, Brisbane, QLD, Australia.

Colleen Niland (C)

Faculty of Medicine, UQ Centre for Clinical Research, The University of Queensland, Herston, Brisbane, QLD, Australia.

Kaltin Ferguson (K)

Faculty of Medicine, UQ Centre for Clinical Research, The University of Queensland, Herston, Brisbane, QLD, Australia.

Irma Gresshoff (I)

Faculty of Medicine, UQ Centre for Clinical Research, The University of Queensland, Herston, Brisbane, QLD, Australia.

Mark Bettington (M)

Faculty of Medicine, UQ Centre for Clinical Research, The University of Queensland, Herston, Brisbane, QLD, Australia.

Nirmala Pathmanathan (N)

Westmead Breast Cancer Institute, University of Sydney, Westmead, NSW, Australia.

Gary M Tse (GM)

Department of Anatomical and Cellular Pathology, Prince of Wales Hospital, The Chinese University of Hong Kong, Sha Tin, Hong Kong.

David Papadimos (D)

Department of Histopathology, Sullivan Nicolaides Pathology, Bowen Hills, QLD, Australia.

Rajadurai Pathmanathan (R)

Jeffrey Cheah School of Medicine & Health Sciences, Monash University Malaysia, 47500, Bandar Sunway, Selangor, Malaysia.

Gavin Harris (G)

Canterbury Health Laboratories, Christchurch, New Zealand/Department of Molecular Medicine and Pathology, University of Auckland, Auckland, New Zealand.

Rin Yamaguchi (R)

Department of Pathology and Laboratory Medicine, Kurume University Medical Center, 155-1 Kokubu, Kurume-shi, 839-0863, Japan.

Puay Hoon Tan (PH)

Division of Pathology, Singapore General Hospital, Singapore, Singapore.

Stephen Fox (S)

Peter MacCallum Cancer Centre and University of Melbourne, Melbourne, VIC, Australia.

Sandra A O'Toole (SA)

Garvan Institute of Medical Research and the Kinghorn Cancer Centre, 384 Victoria Street, Darlinghurst, NSW, 2010, Australia.
Department of Tissue Pathology and Diagnostic Oncology, Royal Prince Alfred Hospital, Camperdown, NSW, 2050, Australia.

Peter T Simpson (PT)

Faculty of Medicine, UQ Centre for Clinical Research, The University of Queensland, Herston, Brisbane, QLD, Australia.

Sunil R Lakhani (SR)

Faculty of Medicine, UQ Centre for Clinical Research, The University of Queensland, Herston, Brisbane, QLD, Australia. s.lakhani@uq.edu.au.
Pathology Queensland, The Royal Brisbane & Women's Hospital, Herston, Brisbane, QLD, Australia. s.lakhani@uq.edu.au.

Amy E McCart Reed (AE)

Faculty of Medicine, UQ Centre for Clinical Research, The University of Queensland, Herston, Brisbane, QLD, Australia. amy.reed@uq.edu.au.
QIMR Berghofer Medical Research Institute, Herston, Brisbane, QLD, Australia. amy.reed@uq.edu.au.

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