Evidence for possible association of vitamin D status with cytokine storm and unregulated inflammation in COVID-19 patients.


Journal

Aging clinical and experimental research
ISSN: 1720-8319
Titre abrégé: Aging Clin Exp Res
Pays: Germany
ID NLM: 101132995

Informations de publication

Date de publication:
Oct 2020
Historique:
received: 20 06 2020
accepted: 04 08 2020
pubmed: 3 9 2020
medline: 21 10 2020
entrez: 3 9 2020
Statut: ppublish

Résumé

We present evidence for a possible role of Vitamin D (VitD) deficiency in unregulated cytokine production and inflammation leading to complications in COVID-19 patients. The time-adjusted case mortality ratio (T-CMR) was estimated as the ratio of deceased patients on day N to the confirmed cases on day N-8. The adaptive average of T-CMR (A-CMR) was calculated as a metric of COVID-19 associated mortality. A model based on positivity change (PC) and an estimated prevalence of COVID-19 was used to determine countries with similar screening strategies. A possible association of A-CMR with the mean concentration of 25-hydroxyvitamin D (25(OH)D) in elderly individuals in countries with similar screening strategy was investigated. We considered high C-reactive protein (CRP) in severe COVID-19 patients (CRP ≥ 1 mg/dL) as a surrogate of a cytokine storm. We considered high-sensitivity CRP (hs-CRP) in healthy subjects as hs-CRP ≥ 0.2 mg/dL. A link between 25(OH)D and A-CMR in countries with similar screening strategy is evidence for VitD's possible role in reducing unregulated cytokine production and inflammation among patients with severe COVID-19. We observed an odds ratio (OR) of 1.8 with 95% confidence interval (95% CI) (1.2 to 2.6) and an OR of 1.9 with 95% CI (1.4 to 2.7) for hs-CRP in VitD deficient elderly from low-income families and high-income families, respectively. COVID-19 patient-level data show an OR of 3.4 with 95% CI (2.15 to 5.4) for high CRP in severe COVID-19 patients. We conclude that future studies on VitD's role in reducing cytokine storm and COVID-19 mortality are warranted.

Identifiants

pubmed: 32876941
doi: 10.1007/s40520-020-01677-y
pii: 10.1007/s40520-020-01677-y
pmc: PMC7465887
doi:

Substances chimiques

Cytokines 0
Vitamin D 1406-16-2
C-Reactive Protein 9007-41-4

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

2141-2158

Subventions

Organisme : NCI NIH HHS
ID : R01 CA225002
Pays : United States

Commentaires et corrections

Type : CommentIn
Type : ErratumIn

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Auteurs

Ali Daneshkhah (A)

Department of Biomedical Engineering, Northwestern University, Evanston, IL, USA.

Vasundhara Agrawal (V)

Department of Biomedical Engineering, Northwestern University, Evanston, IL, USA.

Adam Eshein (A)

Department of Biomedical Engineering, Northwestern University, Evanston, IL, USA.

Hariharan Subramanian (H)

Department of Biomedical Engineering, Northwestern University, Evanston, IL, USA.

Hemant Kumar Roy (HK)

Boston Medical Center, Boston, MA, USA.

Vadim Backman (V)

Department of Biomedical Engineering, Northwestern University, Evanston, IL, USA. v-backman@northwestern.edu.

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