Platelet RNA modules point to coronary calcification in asymptomatic women with former preeclampsia.


Journal

Atherosclerosis
ISSN: 1879-1484
Titre abrégé: Atherosclerosis
Pays: Ireland
ID NLM: 0242543

Informations de publication

Date de publication:
12 2019
Historique:
received: 11 03 2019
revised: 06 09 2019
accepted: 09 10 2019
pubmed: 11 11 2019
medline: 4 8 2020
entrez: 10 11 2019
Statut: ppublish

Résumé

Women who develop preeclampsia during pregnancy are at a higher risk for developing cardiovascular disease. As platelets are affected by preeclampsia, we set out to identify whether platelets carry information in their transcriptome on cardiovascular risk in women with former preeclampsia. Platelets were isolated from asymptomatic women with previous preeclampsia, who underwent screening with coronary computed tomography angiography. Platelet RNA was isolated and used to construct gene networks using an unbiased approach. Platelet gene modules assembled from the network were related to risk factors and clinical traits of these women, including coronary artery calcium scores (CACS). We found multiple gene modules which correlated with CACS (correlation coefficients: 0.44 to 0.59, p = 0.05 to 0.007). The genes from two clinically relevant modules were expressed at a higher level in the group with calcifications (p = 3.9 × 10 We found platelet RNA modules that correlated with CACS in asymptomatic women with previous preeclampsia. Whether or not platelets directly contribute to this disease trajectory, or reflect the underlying plaque substrate remains to be determined, but enrichment for coronary artery disease susceptibility genes emphasizes the importance for the disease.

Sections du résumé

BACKGROUND AND AIMS
Women who develop preeclampsia during pregnancy are at a higher risk for developing cardiovascular disease. As platelets are affected by preeclampsia, we set out to identify whether platelets carry information in their transcriptome on cardiovascular risk in women with former preeclampsia.
METHODS
Platelets were isolated from asymptomatic women with previous preeclampsia, who underwent screening with coronary computed tomography angiography. Platelet RNA was isolated and used to construct gene networks using an unbiased approach. Platelet gene modules assembled from the network were related to risk factors and clinical traits of these women, including coronary artery calcium scores (CACS).
RESULTS
We found multiple gene modules which correlated with CACS (correlation coefficients: 0.44 to 0.59, p = 0.05 to 0.007). The genes from two clinically relevant modules were expressed at a higher level in the group with calcifications (p = 3.9 × 10
CONCLUSIONS
We found platelet RNA modules that correlated with CACS in asymptomatic women with previous preeclampsia. Whether or not platelets directly contribute to this disease trajectory, or reflect the underlying plaque substrate remains to be determined, but enrichment for coronary artery disease susceptibility genes emphasizes the importance for the disease.

Identifiants

pubmed: 31706077
pii: S0021-9150(19)31526-6
doi: 10.1016/j.atherosclerosis.2019.10.009
pii:
doi:

Substances chimiques

RNA 63231-63-0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

114-121

Informations de copyright

Copyright © 2019. Published by Elsevier B.V.

Auteurs

Robin J G Hartman (RJG)

Laboratory of Experimental Cardiology, UMC Utrecht, Utrecht University, Utrecht, the Netherlands.

Suzanne J A Korporaal (SJA)

Laboratory of Experimental Cardiology, UMC Utrecht, Utrecht University, Utrecht, the Netherlands.

Michal Mokry (M)

Laboratory of Experimental Cardiology, UMC Utrecht, Utrecht University, Utrecht, the Netherlands; Department of Pediatric Gastroenterology, Wilhelmina Children's Hospital, Utrecht University, Utrecht, the Netherlands; Laboratory for Clinical Chemistry and Haematology, UMC Utrecht, Utrecht University, Utrecht, the Netherlands.

Saskia C A de Jager (SCA)

Laboratory of Experimental Cardiology, UMC Utrecht, Utrecht University, Utrecht, the Netherlands.

John A L Meeuwsen (JAL)

Laboratory of Experimental Cardiology, UMC Utrecht, Utrecht University, Utrecht, the Netherlands.

Sander W van der Laan (SW)

Laboratory for Clinical Chemistry and Haematology, UMC Utrecht, Utrecht University, Utrecht, the Netherlands.

Nico R Lansu (NR)

Department of Pediatric Gastroenterology, Wilhelmina Children's Hospital, Utrecht University, Utrecht, the Netherlands.

Gerbrand A Zoet (GA)

Department of Obstetrics and Gynecology, UMC Utrecht, Utrecht University, Utrecht, the Netherlands.

Gerard Pasterkamp (G)

Laboratory for Clinical Chemistry and Haematology, UMC Utrecht, Utrecht University, Utrecht, the Netherlands.

Rolf T Urbanus (RT)

Van Creveldkliniek, UMC Utrecht, Utrecht University, Utrecht, the Netherlands.

Imo E Hoefer (IE)

Laboratory for Clinical Chemistry and Haematology, UMC Utrecht, Utrecht University, Utrecht, the Netherlands.

Arie Franx (A)

Department of Obstetrics and Gynecology, UMC Utrecht, Utrecht University, Utrecht, the Netherlands.

Birgitta K Velthuis (BK)

Department of Radiology, UMC Utrecht, Utrecht University, Utrecht, the Netherlands.

Bas B van Rijn (BB)

Department of Obstetrics and Gynecology, UMC Utrecht, Utrecht University, Utrecht, the Netherlands.

Hester M den Ruijter (HM)

Laboratory of Experimental Cardiology, UMC Utrecht, Utrecht University, Utrecht, the Netherlands. Electronic address: h.m.denruijter-2@umcutrecht.nl.

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