ID1 Mediates Escape from TGFβ Tumor Suppression in Pancreatic Cancer.


Journal

Cancer discovery
ISSN: 2159-8290
Titre abrégé: Cancer Discov
Pays: United States
ID NLM: 101561693

Informations de publication

Date de publication:
01 2020
Historique:
received: 07 05 2019
revised: 27 08 2019
accepted: 30 09 2019
pubmed: 5 10 2019
medline: 13 2 2021
entrez: 5 10 2019
Statut: ppublish

Résumé

TGFβ is an important tumor suppressor in pancreatic ductal adenocarcinoma (PDA), yet inactivation of TGFβ pathway components occurs in only half of PDA cases. TGFβ cooperates with oncogenic RAS signaling to trigger epithelial-to-mesenchymal transition (EMT) in premalignant pancreatic epithelial progenitors, which is coupled to apoptosis owing to an imbalance of SOX4 and KLF5 transcription factors. We report that PDAs that develop with the TGFβ pathway intact avert this apoptotic effect via ID1.

Identifiants

pubmed: 31582374
pii: 2159-8290.CD-19-0529
doi: 10.1158/2159-8290.CD-19-0529
pmc: PMC6954299
mid: NIHMS1541060
doi:

Substances chimiques

Biomarkers, Tumor 0
ID1 protein, human 0
Inhibitor of Differentiation Protein 1 0
Transforming Growth Factor beta 0

Types de publication

Journal Article Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

142-157

Subventions

Organisme : NCI NIH HHS
ID : T32 CA160001
Pays : United States
Organisme : NCI NIH HHS
ID : R35 CA220508
Pays : United States
Organisme : NCI NIH HHS
ID : P30 CA008748
Pays : United States
Organisme : NCI NIH HHS
ID : R01 CA034610
Pays : United States
Organisme : NIGMS NIH HHS
ID : T32 GM007739
Pays : United States
Organisme : NCI NIH HHS
ID : F30 CA203238
Pays : United States

Informations de copyright

©2019 American Association for Cancer Research.

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Auteurs

Yun-Han Huang (YH)

Cancer Biology and Genetics Program, Sloan Kettering Institute, Memorial Sloan Kettering Cancer Center, New York, New York.
Weill Cornell/Sloan Kettering/Rockefeller Tri-Institutional MD-PhD Program, New York, New York.
Gerstner Sloan Kettering Graduate School of Biomedical Sciences, New York, New York.

Jing Hu (J)

Cancer Biology and Genetics Program, Sloan Kettering Institute, Memorial Sloan Kettering Cancer Center, New York, New York.

Fei Chen (F)

Cancer Biology and Genetics Program, Sloan Kettering Institute, Memorial Sloan Kettering Cancer Center, New York, New York.

Nicolas Lecomte (N)

The David M. Rubinstein Center for Pancreatic Cancer Research, Memorial Sloan Kettering Cancer Center, New York, New York.

Harihar Basnet (H)

Cancer Biology and Genetics Program, Sloan Kettering Institute, Memorial Sloan Kettering Cancer Center, New York, New York.

Charles J David (CJ)

Cancer Biology and Genetics Program, Sloan Kettering Institute, Memorial Sloan Kettering Cancer Center, New York, New York.

Matthew D Witkin (MD)

Center for Epigenetics Research, Memorial Sloan Kettering Cancer Center, New York, New York.

Peter J Allen (PJ)

Department of Surgery, Memorial Sloan Kettering Cancer Center, New York, New York.

Steven D Leach (SD)

The David M. Rubinstein Center for Pancreatic Cancer Research, Memorial Sloan Kettering Cancer Center, New York, New York.
Department of Surgery, Memorial Sloan Kettering Cancer Center, New York, New York.
Department of Pathology, Memorial Sloan Kettering Cancer Center, New York, New York.

Travis J Hollmann (TJ)

Department of Pathology, Memorial Sloan Kettering Cancer Center, New York, New York.
Human Oncology and Pathogenesis Program, Memorial Sloan Kettering Cancer Center, New York, New York.

Christine A Iacobuzio-Donahue (CA)

The David M. Rubinstein Center for Pancreatic Cancer Research, Memorial Sloan Kettering Cancer Center, New York, New York.
Department of Pathology, Memorial Sloan Kettering Cancer Center, New York, New York.
Human Oncology and Pathogenesis Program, Memorial Sloan Kettering Cancer Center, New York, New York.

Joan Massagué (J)

Cancer Biology and Genetics Program, Sloan Kettering Institute, Memorial Sloan Kettering Cancer Center, New York, New York. j-massague@ski.mskcc.org.

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