Resveratrol treatment in patients with polycystic ovary syndrome decreased pro-inflammatory and endoplasmic reticulum stress markers.


Journal

American journal of reproductive immunology (New York, N.Y. : 1989)
ISSN: 1600-0897
Titre abrégé: Am J Reprod Immunol
Pays: Denmark
ID NLM: 8912860

Informations de publication

Date de publication:
01 2020
Historique:
received: 28 08 2018
revised: 17 07 2019
accepted: 21 08 2019
pubmed: 5 9 2019
medline: 1 12 2020
entrez: 5 9 2019
Statut: ppublish

Résumé

Polycystic ovary syndrome (PCOS) is associated with endoplasmic reticulum (ER) stress and pro-inflammatory condition. The aim of the present study was to evaluate the effect of resveratrol treatment on pro-inflammatory and ER stress markers in patients with PCOS. Cumulus cells were obtained from 40 patients with PCOS who were divided into two groups: placebo and resveratrol treatment (receiving 800 mg/d for 40 days) groups. Blood samples were obtained from all patients before and after the procedure to evaluate interleukin (IL)-6, IL-1β, IL-18, TNF-α, NF-κB, and C-reactive protein (CRP). Total RNA was extracted from cumulus cells, and cDNA was synthesized by reverse transcription. Expressions of five genes in ER stress response pathway (ATF4, ATF6, CHOP, GRP78, and XBP1s) were assessed with quantitative real-time PCR. Statistical analysis was performed with Student's t test. After treatment with resveratrol, it was found that serum levels of IL-6, IL-1β, TNF-α, IL-18, NF-κB, and CRP decreased in the treatment group. In addition, gene expression results showed that the expression levels of ATF4 (P < .05) and ATF6 (P < .001) significantly increased in the resveratrol treatment group, while the expression levels of CHOP, GRP78, and XBP1 (P < .001 for all) significantly decreased. Results demonstrated that resveratrol has anti-inflammatory effects through the suppression of NF-κB and NF-κB-regulated gene products. On the other hand, resveratrol can modulate ER stress in granulosa cells (GCs) by altering the expression of genes involved in unfolding protein response (UPR) process. Our findings suggest that ER stress is a potential therapeutic target for patients with PCOS.

Identifiants

pubmed: 31483910
doi: 10.1111/aji.13186
doi:

Substances chimiques

ATF4 protein, human 0
ATF6 protein, human 0
Activating Transcription Factor 6 0
Anti-Inflammatory Agents 0
Cytokines 0
DDIT3 protein, human 0
Endoplasmic Reticulum Chaperone BiP 0
HSPA5 protein, human 0
Heat-Shock Proteins 0
NF-kappa B 0
X-Box Binding Protein 1 0
XBP1 protein, human 0
Activating Transcription Factor 4 145891-90-3
Transcription Factor CHOP 147336-12-7
C-Reactive Protein 9007-41-4
Resveratrol Q369O8926L

Types de publication

Journal Article Randomized Controlled Trial Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

e13186

Subventions

Organisme : Tehran University of Medical Sciences and Health Services
ID : 94-03-30-29803
Pays : International

Informations de copyright

© 2019 John Wiley & Sons A/S. Published by John Wiley & Sons Ltd.

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Auteurs

Samaneh Brenjian (S)

Department of Anatomy, School of Medicine, Tehran University of Medical Sciences, Tehran, Iran.

Ashraf Moini (A)

Department of Gynecology and Obstetrics, School of Medicine, Tehran University of Medical Science, Tehran, Iran.
Department of Endocrinology and Female Infertility, Reproductive Biomedicine Research Center, Royan Institute for Reproductive Biomedicine, ACECR, Tehran, Iran.
Vali-e-Asr Reproductive Health Research Center, Tehran University of Medical Sciences, Tehran, Iran.

Nazila Yamini (N)

Embryology Laboratory, Department of ART, Arash Women's Hospital, Tehran University of Medical Science, Tehran, Iran.

Ladan Kashani (L)

Department of Gynecology and Obstetrics, School of Medicine, Tehran University of Medical Science, Tehran, Iran.

Maryam Faridmojtahedi (M)

Department of Gynecology and Obstetrics, School of Medicine, Tehran University of Medical Science, Tehran, Iran.

Mojdeh Bahramrezaie (M)

Department of Anatomy, School of Medicine, Tehran University of Medical Sciences, Tehran, Iran.

Mahshad Khodarahmian (M)

Department of Anatomy, School of Medicine, Tehran University of Medical Sciences, Tehran, Iran.

Fardin Amidi (F)

Department of Anatomy, School of Medicine, Tehran University of Medical Sciences, Tehran, Iran.
Department of Infertility, Shariati Hospital, Tehran University of Medical Sciences, Tehran, Iran.

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