Switching renal transplant recipients to belatacept therapy: results of a real-life gradual conversion protocol.


Journal

Transplant immunology
ISSN: 1878-5492
Titre abrégé: Transpl Immunol
Pays: Netherlands
ID NLM: 9309923

Informations de publication

Date de publication:
10 2019
Historique:
received: 22 02 2019
revised: 18 04 2019
accepted: 26 04 2019
pubmed: 10 5 2019
medline: 4 4 2020
entrez: 10 5 2019
Statut: ppublish

Résumé

Conversion to belatacept immunosuppression is a therapeutic option for renal-transplant recipients with calcineurin inhibitors (CNI) toxicity, but it associates with high risk of acute rejection. Gradual conversion and serial immune monitoring with urinary chemokine CXCL9 may allow increasing safety of this maneuver. We converted kidney transplant recipients with signs of toxicity to CNI or other immunosuppressive drugs to belatacept over a 2-month period. We monitored renal function, metabolic profile, and circulating lymphocyte subsets. We also quantified urinary CXCL9 over a 12-month follow-up period. Between September 2016 and March 2017, 35 patients were successfully switched to belatacept immunosuppression at 3.3 (1.3-7.2) years after transplant. Two patients had a reversible rise in serum creatinine, associated with acute rejection in one case. Urinary CXCL9 increased before serum creatinine. After conversion, blood pressure and HbA1c significantly declined while eGFR and proteinuria remained stable. The percentage of circulating effector T cells and memory B cells significantly declined. Conversion from CNI to belatacept, in this setting, was feasible and safe, provided it was performed over a 2-month time-period. Monitoring urinary CXCL9 may further increase safety through earlier identification of patients at risk for acute rejection. The procedure associates with improved blood pressure, metabolic profile, and reduced circulating effector T and B cells.

Identifiants

pubmed: 31071442
pii: S0966-3274(19)30023-1
doi: 10.1016/j.trim.2019.04.002
pii:
doi:

Substances chimiques

Biomarkers 0
CXCL9 protein, human 0
Calcineurin Inhibitors 0
Chemokine CXCL9 0
Immunosuppressive Agents 0
Abatacept 7D0YB67S97

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

101207

Informations de copyright

Copyright © 2019 Elsevier B.V. All rights reserved.

Auteurs

Paolo Malvezzi (P)

Service de Néphrologie, Hémodialyse, Aphérèses et Transplantation, CHU Grenoble Alpes, Grenoble, France. Electronic address: pmalvezzi@chu-grenoble.fr.

Clara Fischman (C)

Department of Medicine, Icahn School of Medicine at Mount Sinai, New York, NY, United States.

Guillaume Rigault (G)

Service de Néphrologie, Hémodialyse, Aphérèses et Transplantation, CHU Grenoble Alpes, Grenoble, France.

Marie Christine Jacob (MC)

Departement d'Immunologie, CHU Grenoble Alpes, Grenoble, France.

Tatiana Raskovalova (T)

Departement d'Immunologie, CHU Grenoble Alpes, Grenoble, France.

Thomas Jouve (T)

Service de Néphrologie, Hémodialyse, Aphérèses et Transplantation, CHU Grenoble Alpes, Grenoble, France; Université Grenoble Alpes, Grenoble, France.

Bénédicte Janbon (B)

Service de Néphrologie, Hémodialyse, Aphérèses et Transplantation, CHU Grenoble Alpes, Grenoble, France.

Lionel Rostaing (L)

Service de Néphrologie, Hémodialyse, Aphérèses et Transplantation, CHU Grenoble Alpes, Grenoble, France; Université Grenoble Alpes, Grenoble, France.

Paolo Cravedi (P)

Department of Medicine, Icahn School of Medicine at Mount Sinai, New York, NY, United States.

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Classifications MeSH