Stroke and antiphospholipid syndrome-antiphospholipid antibodies are a risk factor for an ischemic cerebrovascular event.


Journal

Clinical rheumatology
ISSN: 1434-9949
Titre abrégé: Clin Rheumatol
Pays: Germany
ID NLM: 8211469

Informations de publication

Date de publication:
Feb 2019
Historique:
received: 21 04 2018
accepted: 27 07 2018
revised: 26 06 2018
pubmed: 9 8 2018
medline: 29 5 2019
entrez: 9 8 2018
Statut: ppublish

Résumé

Testing for antiphospholipid antibodies could be an important part in determining the cause of a cerebrovascular event (CVE). Currently, it is also unknown whether antiphospholipid antibodies represent a risk factor for the development of a CVE and whether the selected therapy options are efficacious. So, this study aimed at (1) determining the frequency of patients experiencing a CVE and fulfilling the laboratory criterion for an antiphospholipid syndrome (APS), (2) investigating whether the persistent presence of antiphospholipid antibodies represented a risk factor for a CVE, and (3) focusing on the efficacy of the selected treatment strategy in the first year after the CVE. Eighty-nine patients with an acute CVE were prospectively followed for 1 year. At least two sera from each were tested for lupus anticoagulants, anticardiolipin, anti-β2-glycoprotein I, anti-phosphatidylserine/prothrombin and anti-annexin V antibodies. Twenty out of eighty-nine (22%) of CVE patients fulfilled the criteria for APS (17/20 for definitive and 3 for probable APS). There was a significant association between persistently present antiphospholipid antibodies and the CVE (OR, 4.62). No statistically significant difference was found in the CVE recurrence rate between APS-CVE and non-APS-CVE patients being treated mainly with acetyl salicylic acid. Antiphospholipid antibodies represent an independent risk factor for a CVE. In the first year after the CVE, antiplatelet therapy seemed to be sufficient in secondary CVE thromboprophylaxis in most APS patients.

Identifiants

pubmed: 30088114
doi: 10.1007/s10067-018-4247-3
pii: 10.1007/s10067-018-4247-3
doi:

Substances chimiques

Antibodies, Antiphospholipid 0
Anticoagulants 0
Lupus Coagulation Inhibitor 0
beta 2-Glycoprotein I 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

379-384

Subventions

Organisme : Javna Agencija za Raziskovalno Dejavnost RS
ID : P3-0314

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Auteurs

Nataša Gašperšič (N)

Department of Rheumatology, University Medical Centre Ljubljana, Vodnikova 62, SI-1000, Ljubljana, Slovenia. natasa.gaspersic@kclj.si.

Marjan Zaletel (M)

Department of Neurology, University Medical Centre Ljubljana, Zaloška 2, SI-1000, Ljubljana, Slovenia.

Jan Kobal (J)

Department of Neurology, University Medical Centre Ljubljana, Zaloška 2, SI-1000, Ljubljana, Slovenia.

Polona Žigon (P)

Department of Rheumatology, University Medical Centre Ljubljana, Vodnikova 62, SI-1000, Ljubljana, Slovenia.

Saša Čučnik (S)

Department of Rheumatology, University Medical Centre Ljubljana, Vodnikova 62, SI-1000, Ljubljana, Slovenia.
Faculty of Pharmacy, University of Ljubljana, Aškerčeva 7, SI-1000, Ljubljana, Slovenia.

Snežna Sodin Šemrl (SS)

Department of Rheumatology, University Medical Centre Ljubljana, Vodnikova 62, SI-1000, Ljubljana, Slovenia.
Faculty of Mathematics, Natural Sciences and Information Technologies, University of Primorska, Glagoljaška 8, SI-6000, Koper, Slovenia.

Matija Tomšič (M)

Department of Rheumatology, University Medical Centre Ljubljana, Vodnikova 62, SI-1000, Ljubljana, Slovenia.
Faculty of Medicine, University of Ljubljana, Vrazov trg 2, SI-1104, Ljubljana, Slovenia.

Aleš Ambrožič (A)

Department of Rheumatology, University Medical Centre Ljubljana, Vodnikova 62, SI-1000, Ljubljana, Slovenia.

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