Jana F Tegethoff

Kevin Roehle 1 collaboration

Li Qiang 1 collaboration

Katherine S Ventre 1 collaboration

Daniel Heid 1 collaboration

Lestat R Ali 1 collaboration

Patrick Lenehan 1 collaboration

Max Heckler 1 collaboration

Stephanie J Crowley 1 collaboration

Courtney T Stump 1 collaboration

Gabrielle Ro 1 collaboration

Anže Godicelj 1 collaboration

Aladdin M Bhuiyan 1 collaboration

Annan Yang 1 collaboration

Maria Quiles Del Rey 1 collaboration

Tamara Biary 1 collaboration

Adrienne M Luoma 1 collaboration

Patrick T Bruck 1 collaboration

Svenja L Nopper 1 collaboration

Jinyang Li 1 collaboration

Katelyn T Byrne 1 collaboration

Marc Pelletier 1 collaboration

Kai W Wucherpfennig 1 collaboration

Ben Z Stanger 1 collaboration

James J Akin 1 collaboration

Joseph D Mancias 1 collaboration

Judith Agudo 1 collaboration

Michael Dougan 1 collaboration

Stephanie K Dougan 1 collaboration

Hamid Dolatshad 1 collaboration

Dharamveer Tatwavedi 1 collaboration

Doaa Ahmed 1 collaboration

Jacqueline Boultwood 1 collaboration

Andrea Pellagatti 1 collaboration

Bettina Weigel 1 collaboration

Sarah D Grieder 1 collaboration

Bryce Lim 1 collaboration

Bhuvaneswari Nagarajan 1 collaboration

Yu-Chao Liu 1 collaboration

Jule Truberg 1 collaboration

Dimitris Papageorgiou 1 collaboration

Juan M Adrian-Segarra 1 collaboration

Laura K Schmidt 1 collaboration

Janina Kaspar 1 collaboration

Eric Poisel 1 collaboration

Elisa Heinzelmann 1 collaboration

Manu Saraswat 1 collaboration

Marleen Christ 1 collaboration

Christian Arnold 1 collaboration

Ignacio L Ibarra 1 collaboration

Joaquin Campos 1 collaboration

Jeroen Krijgsveld 1 collaboration

Hannah Monyer 1 collaboration

Judith B Zaugg 1 collaboration

Claudio Acuna 1 collaboration

Moritz Mall 1 collaboration

Affiliations

Publications (3)

cIAP1/2 antagonism eliminates MHC class I-negative tumors through T cell-dependent reprogramming of mononuclear phagocytes.

Application of induced pluripotent stem cell technology for the investigation of hematological disorders.

MYT1L haploinsufficiency in human neurons and mice causes autism-associated phenotypes that can be reversed by genetic and pharmacologic intervention.

Réseau de co-auteurs