Case series: Downbeat nystagmus in SCA27B.


Journal

Journal of the neurological sciences
ISSN: 1878-5883
Titre abrégé: J Neurol Sci
Pays: Netherlands
ID NLM: 0375403

Informations de publication

Date de publication:
15 Nov 2023
Historique:
received: 26 08 2023
revised: 16 10 2023
accepted: 22 10 2023
medline: 27 11 2023
pubmed: 1 11 2023
entrez: 31 10 2023
Statut: ppublish

Résumé

Spinocerebellar ataxia (SCA) 27B, first reported in late 2022, is caused by the abnormal expansion of GAA repeats in the first intron of the FGF14 gene, which encodes the fibroblast growth factor 14. We present two late-onset cases, each manifesting mild cerebellar ataxia accompanied by omnidirectional downbeat nystagmus, which was enhanced in a suspended head position. None of the patients exhibited impaired head impulse or caloric tests. Repeat-primed PCR and targeted long-read nanopore sequence analysis of the FGF14 GAA repeat site identified more than 250 repeats, leading to the diagnosis of SCA27B. Downbeat nystagmus is reported to be associated with disturbances in the suppression of the vestibulo-ocular reflex (VOR). Our patients with SCA27B demonstrated downbeat nystagmus, likely due to a disruption of the VOR at the level of the cerebellar cortex, a potentially characteristic clinical feature of SCA27B. We have included video footages of eye movements recorded using Frenzel goggles for these cases. Omnidirectional downbeat nystagmus may be a distinctive clinical feature of SCA27B.

Sections du résumé

BACKGROUND BACKGROUND
Spinocerebellar ataxia (SCA) 27B, first reported in late 2022, is caused by the abnormal expansion of GAA repeats in the first intron of the FGF14 gene, which encodes the fibroblast growth factor 14.
CASE PRESENTATION METHODS
We present two late-onset cases, each manifesting mild cerebellar ataxia accompanied by omnidirectional downbeat nystagmus, which was enhanced in a suspended head position. None of the patients exhibited impaired head impulse or caloric tests. Repeat-primed PCR and targeted long-read nanopore sequence analysis of the FGF14 GAA repeat site identified more than 250 repeats, leading to the diagnosis of SCA27B.
DISCUSSION CONCLUSIONS
Downbeat nystagmus is reported to be associated with disturbances in the suppression of the vestibulo-ocular reflex (VOR). Our patients with SCA27B demonstrated downbeat nystagmus, likely due to a disruption of the VOR at the level of the cerebellar cortex, a potentially characteristic clinical feature of SCA27B. We have included video footages of eye movements recorded using Frenzel goggles for these cases.
CONCLUSIONS CONCLUSIONS
Omnidirectional downbeat nystagmus may be a distinctive clinical feature of SCA27B.

Identifiants

pubmed: 37907039
pii: S0022-510X(23)00310-6
doi: 10.1016/j.jns.2023.120849
pii:
doi:

Types de publication

Case Reports Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

120849

Informations de copyright

Copyright © 2023. Published by Elsevier B.V.

Déclaration de conflit d'intérêts

Declaration of Competing Interest The authors report no relevant disclosures. Full disclosure from information provided by the authors is available with the full text of this article.

Auteurs

Shinichi Shirai (S)

Departments of Neurology, Hokkaido University, Sapporo, Japan.

Keiichi Mizushima (K)

Departments of Neurology, Hokkaido University, Sapporo, Japan.

Keishi Fujiwara (K)

Otolaryngology-Head & Neck Surgery, Faculty of Medicine and Graduate School of Medicine, Hokkaido University, Sapporo, Japan.

Eriko Koshimizu (E)

Department of Human Genetics, Yokohama City University Graduate School of Medicine, Yokohama, Japan.

Masaaki Matsushima (M)

Departments of Neurology, Hokkaido University, Sapporo, Japan.

Satoko Miyatake (S)

Department of Human Genetics, Yokohama City University Graduate School of Medicine, Yokohama, Japan; Department of Clinical Genetics, Yokohama City University Hospital, Yokohama, Japan.

Ikuko Iwata (I)

Departments of Neurology, Hokkaido University, Sapporo, Japan.

Hiroaki Yaguchi (H)

Departments of Neurology, Hokkaido University, Sapporo, Japan.

Naomichi Matsumoto (N)

Department of Human Genetics, Yokohama City University Graduate School of Medicine, Yokohama, Japan.

Ichiro Yabe (I)

Departments of Neurology, Hokkaido University, Sapporo, Japan. Electronic address: yabe@med.hokudai.ac.jp.

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