Tamoxifen-predictive value of gene expression signatures in premenopausal breast cancer: data from the randomized SBII:2 trial.


Journal

Breast cancer research : BCR
ISSN: 1465-542X
Titre abrégé: Breast Cancer Res
Pays: England
ID NLM: 100927353

Informations de publication

Date de publication:
29 09 2023
Historique:
received: 28 06 2023
accepted: 25 09 2023
medline: 2 10 2023
pubmed: 29 9 2023
entrez: 29 9 2023
Statut: epublish

Résumé

Gene expression (GEX) signatures in breast cancer provide prognostic information, but little is known about their predictive value for tamoxifen treatment. We examined the tamoxifen-predictive value and prognostic effects of different GEX signatures in premenopausal women with early breast cancer. RNA from formalin-fixed paraffin-embedded tumor tissue from premenopausal women randomized between two years of tamoxifen treatment and no systemic treatment was extracted and successfully subjected to GEX profiling (n = 437, NanoString Breast Cancer 360™ panel). The median follow-up periods for a recurrence-free interval (RFi) and overall survival (OS) were 28 and 33 years, respectively. Associations between GEX signatures and tamoxifen effect were assessed in patients with estrogen receptor-positive/human epidermal growth factor receptor 2-negative (ER+ /HER2-) tumors using Kaplan-Meier estimates and Cox regression. The prognostic effects of GEX signatures were studied in the entire cohort. False discovery rate adjustments (q-values) were applied to account for multiple hypothesis testing. In patients with ER+/HER2- tumors, FOXA1 expression below the median was associated with an improved effect of tamoxifen after 10 years with regard to RFi (hazard ratio [HR] Expression of FOXA1 is a promising predictive biomarker for tamoxifen effect in ER+/HER2- premenopausal breast cancer. In addition, each of the signatures BC proliferation, Hypoxia, Mast cells, and the GEX of AR, ESR1, and PGR had prognostic value, also after adjusting for established prognostic factors. Trial registration This trial was retrospectively registered in the ISRCTN database the 6th of December 2019, trial ID: https://clinicaltrials.gov/ct2/show/ISRCTN12474687 .

Sections du résumé

BACKGROUND
Gene expression (GEX) signatures in breast cancer provide prognostic information, but little is known about their predictive value for tamoxifen treatment. We examined the tamoxifen-predictive value and prognostic effects of different GEX signatures in premenopausal women with early breast cancer.
METHODS
RNA from formalin-fixed paraffin-embedded tumor tissue from premenopausal women randomized between two years of tamoxifen treatment and no systemic treatment was extracted and successfully subjected to GEX profiling (n = 437, NanoString Breast Cancer 360™ panel). The median follow-up periods for a recurrence-free interval (RFi) and overall survival (OS) were 28 and 33 years, respectively. Associations between GEX signatures and tamoxifen effect were assessed in patients with estrogen receptor-positive/human epidermal growth factor receptor 2-negative (ER+ /HER2-) tumors using Kaplan-Meier estimates and Cox regression. The prognostic effects of GEX signatures were studied in the entire cohort. False discovery rate adjustments (q-values) were applied to account for multiple hypothesis testing.
RESULTS
In patients with ER+/HER2- tumors, FOXA1 expression below the median was associated with an improved effect of tamoxifen after 10 years with regard to RFi (hazard ratio [HR]
CONCLUSIONS
Expression of FOXA1 is a promising predictive biomarker for tamoxifen effect in ER+/HER2- premenopausal breast cancer. In addition, each of the signatures BC proliferation, Hypoxia, Mast cells, and the GEX of AR, ESR1, and PGR had prognostic value, also after adjusting for established prognostic factors. Trial registration This trial was retrospectively registered in the ISRCTN database the 6th of December 2019, trial ID: https://clinicaltrials.gov/ct2/show/ISRCTN12474687 .

Identifiants

pubmed: 37773134
doi: 10.1186/s13058-023-01719-z
pii: 10.1186/s13058-023-01719-z
pmc: PMC10540453
doi:

Substances chimiques

Tamoxifen 094ZI81Y45
Antineoplastic Agents, Hormonal 0

Banques de données

ISRCTN
['ISRCTN12474687']

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

110

Informations de copyright

© 2023. BioMed Central Ltd., part of Springer Nature.

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Auteurs

Christine Lundgren (C)

Department of Oncology, Region Jönköping County, Jönköping, Sweden. christine.lundgren@rjl.se.
Department of Biomedical and Clinical Sciences, Linköping University, Linköping, Sweden. christine.lundgren@rjl.se.
Division of Oncology, Department of Clinical Sciences Lund, Lund University, Medicon Village, Building 404, 223 81, Lund, Sweden. christine.lundgren@rjl.se.

Julia Tutzauer (J)

Division of Oncology, Department of Clinical Sciences Lund, Lund University, Medicon Village, Building 404, 223 81, Lund, Sweden.

Sarah E Church (SE)

NanoString Technologies, Inc., Seattle, USA.

Olle Stål (O)

Department of Biomedical and Clinical Sciences, Linköping University, Linköping, Sweden.

Maria Ekholm (M)

Department of Oncology, Region Jönköping County, Jönköping, Sweden.
Department of Biomedical and Clinical Sciences, Linköping University, Linköping, Sweden.

Carina Forsare (C)

Division of Oncology, Department of Clinical Sciences Lund, Lund University, Medicon Village, Building 404, 223 81, Lund, Sweden.

Bo Nordenskjöld (B)

Department of Biomedical and Clinical Sciences, Linköping University, Linköping, Sweden.

Mårten Fernö (M)

Division of Oncology, Department of Clinical Sciences Lund, Lund University, Medicon Village, Building 404, 223 81, Lund, Sweden.

Pär-Ola Bendahl (PO)

Division of Oncology, Department of Clinical Sciences Lund, Lund University, Medicon Village, Building 404, 223 81, Lund, Sweden.

Lisa Rydén (L)

Division of Surgery, Department of Clinical Sciences Lund, Lund University, Lund, Sweden.
Department of Surgery, Skåne University Hospital, Malmö, Sweden.

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