Cardiorenal Disease in COVID-19 Patients.
Journal
Journal of the renin-angiotensin-aldosterone system : JRAAS
ISSN: 1752-8976
Titre abrégé: J Renin Angiotensin Aldosterone Syst
Pays: England
ID NLM: 100971636
Informations de publication
Date de publication:
2022
2022
Historique:
received:
20
01
2022
revised:
28
02
2022
accepted:
09
03
2022
entrez:
1
4
2022
pubmed:
2
4
2022
medline:
5
4
2022
Statut:
epublish
Résumé
Coronavirus disease 2019 (COVID-19) is an illness caused by a novel coronavirus called severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). Mutations in the genetic coding and the variations in the spike proteins are critical for the virus's mechanism of facilitating fusion with the human host, making the disease more severe. Recent research indicates that comorbidities including diabetes, hypertension, renal disease, heart failure, and atherosclerosis play a significant role in the severity and high mortality rates of (COVID-19), suggesting that perhaps the metabolic syndrome and its components are associated with COVID-19 morbidity. Primarily, angiotensin-converting enzyme 2 (ACE2) receptor is identified as the entrance receptor of SARS-CoV-2. Increased ACE2 expression, endothelial dysfunction plays a vital role in the progression and severity of complications developed due to COVID-19. In this review, we will discuss the association and management of cardiorenal disease and COVID-19.
Identifiants
pubmed: 35359461
doi: 10.1155/2022/4640788
pmc: PMC8956393
doi:
Substances chimiques
Peptidyl-Dipeptidase A
EC 3.4.15.1
Types de publication
Journal Article
Review
Langues
eng
Sous-ensembles de citation
IM
Pagination
4640788Informations de copyright
Copyright © 2022 Muhtasham Sifaat et al.
Déclaration de conflit d'intérêts
The author declares no conflicts of interest in this work.
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