The prognostic value of cardiac troponin for 60 day mortality and major adverse events in COVID-19 patients.


Journal

Cardiovascular pathology : the official journal of the Society for Cardiovascular Pathology
ISSN: 1879-1336
Titre abrégé: Cardiovasc Pathol
Pays: United States
ID NLM: 9212060

Informations de publication

Date de publication:
Historique:
received: 05 05 2021
revised: 02 07 2021
accepted: 27 07 2021
pubmed: 7 8 2021
medline: 8 10 2021
entrez: 6 8 2021
Statut: ppublish

Résumé

The variability of coronavirus disease 2019 (COVID-19) illness severity has puzzled clinicians and has sparked efforts to better predict who would benefit from rapid intervention. One promising biomarker for in-hospital morbidity and mortality is cardiac troponin (cTn). A retrospective study of 1331 adult patients with COVID-19 admitted to the Rush University System in Illinois, USA was performed. Patients without cTn measurement during their admission or a history of end stage renal disease or stage 5 chronic kidney disease were excluded. Using logistic regression adjusted for baseline characteristics, pre-existing comorbidities, and other laboratory markers of inflammation, cTn was assessed as a predictor of 60-day mortality and severe COVID-19 infection, consisting of a composite of 60-day mortality, need for intensive care unit, or requiring non-invasive positive pressure ventilation or intubation. A total of 772 patients met inclusion criteria. Of these, 69 (8.9%) had mild cTn elevation (> 1 to < 2x upper limit of normal (ULN)) and 46 (6.0%) had severe cTn elevation (≥ 2x ULN). Regardless of baseline characteristics, comorbidities, and initial c-reactive protein, lactate dehydrogenase, and ferritin, when compared to the normal cTn group, mild cTn elevation and severe cTn elevation were predictors of severe COVID-19 infection (adjusted OR [aOR] aOR 3.00 [CI: 1.51 - 6.29], P < 0.01; aOR 9.96 [CI: 2.75 - 64.23], P < 0.01, respectively); severe cTn elevation was a predictor of in-hospital mortality (aOR 2.42 [CI: 1.10 - 5.21], P < 0.05) and 60-day mortality (aOR 2.45 [CI: 1.13 - 5.25], P < 0.05). In our cohort, both mild and severe initial cTn elevation were predictors of severe COVID-19 infection, while only severe cTn elevation was predictive of 60-day mortality. First cTn value on hospitalization is a valuable longitudinal prognosticator for COVID-19 disease severity and mortality.

Sections du résumé

BACKGROUND BACKGROUND
The variability of coronavirus disease 2019 (COVID-19) illness severity has puzzled clinicians and has sparked efforts to better predict who would benefit from rapid intervention. One promising biomarker for in-hospital morbidity and mortality is cardiac troponin (cTn).
METHODS METHODS
A retrospective study of 1331 adult patients with COVID-19 admitted to the Rush University System in Illinois, USA was performed. Patients without cTn measurement during their admission or a history of end stage renal disease or stage 5 chronic kidney disease were excluded. Using logistic regression adjusted for baseline characteristics, pre-existing comorbidities, and other laboratory markers of inflammation, cTn was assessed as a predictor of 60-day mortality and severe COVID-19 infection, consisting of a composite of 60-day mortality, need for intensive care unit, or requiring non-invasive positive pressure ventilation or intubation.
RESULTS RESULTS
A total of 772 patients met inclusion criteria. Of these, 69 (8.9%) had mild cTn elevation (> 1 to < 2x upper limit of normal (ULN)) and 46 (6.0%) had severe cTn elevation (≥ 2x ULN). Regardless of baseline characteristics, comorbidities, and initial c-reactive protein, lactate dehydrogenase, and ferritin, when compared to the normal cTn group, mild cTn elevation and severe cTn elevation were predictors of severe COVID-19 infection (adjusted OR [aOR] aOR 3.00 [CI: 1.51 - 6.29], P < 0.01; aOR 9.96 [CI: 2.75 - 64.23], P < 0.01, respectively); severe cTn elevation was a predictor of in-hospital mortality (aOR 2.42 [CI: 1.10 - 5.21], P < 0.05) and 60-day mortality (aOR 2.45 [CI: 1.13 - 5.25], P < 0.05).
CONCLUSION CONCLUSIONS
In our cohort, both mild and severe initial cTn elevation were predictors of severe COVID-19 infection, while only severe cTn elevation was predictive of 60-day mortality. First cTn value on hospitalization is a valuable longitudinal prognosticator for COVID-19 disease severity and mortality.

Identifiants

pubmed: 34358679
pii: S1054-8807(21)00058-2
doi: 10.1016/j.carpath.2021.107374
pmc: PMC8330143
pii:
doi:

Substances chimiques

Biomarkers 0
Troponin 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

107374

Informations de copyright

Copyright © 2021. Published by Elsevier Inc.

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Auteurs

Max Ruge (M)

Department of Internal Medicine, Thomas Jefferson University Hospital, Philadelphia, Pennsylvania. Electronic address: Max.Ruge@jefferson.edu.

Joanne Michelle D Gomez (JMD)

Division of Cardiology, Rush University Medical Center, Chicago, Illinois.

Jeanne du Fay de Lavallaz (J)

Department of Internal Medicine, Rush University Medical Center, Chicago, Illinois.

Alexander Hlepas (A)

Department of Internal Medicine, Rush University Medical Center, Chicago, Illinois.

Annas Rahman (A)

Department of Internal Medicine, Rush University Medical Center, Chicago, Illinois.

Priya Patel (P)

Department of Internal Medicine, Rush University Medical Center, Chicago, Illinois.

Prutha Lavani (P)

Division of Cardiology, Rush University Medical Center, Chicago, Illinois.

Gatha G Nair (GG)

Division of Cardiology, Rush University Medical Center, Chicago, Illinois.

Nusrat Jahan (N)

Division of Cardiology, Rush University Medical Center, Chicago, Illinois.

J Alan Simmons (JA)

Research Core, Rush University Medical Center, Chicago, Illinois.

Anupama K Rao (AK)

Division of Cardiology, Rush University Medical Center, Chicago, Illinois.

Kim A Williams (KA)

Division of Cardiology, Rush University Medical Center, Chicago, Illinois.

Annabelle Santos Volgman (AS)

Division of Cardiology, Rush University Medical Center, Chicago, Illinois.

Karolina Marinescu (K)

Division of Cardiology, Rush University Medical Center, Chicago, Illinois.

Tisha Suboc (T)

Division of Cardiology, Rush University Medical Center, Chicago, Illinois.

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Classifications MeSH