Chiral deaza-coelenterazine analogs for probing a substrate-binding site in the Ca2+-binding photoprotein aequorin.


Journal

PloS one
ISSN: 1932-6203
Titre abrégé: PLoS One
Pays: United States
ID NLM: 101285081

Informations de publication

Date de publication:
2021
Historique:
received: 07 04 2021
accepted: 01 05 2021
entrez: 11 6 2021
pubmed: 12 6 2021
medline: 25 2 2023
Statut: epublish

Résumé

The Ca2+-binding photoprotein aequorin is a complex of apoAequorin (apoprotein) and (S)-2-peroxycoelenterazine. Aequorin can be regenerated by the incubation of apoAequorin with coelenterazine and molecular oxygen (O2). In this study, to investigate the molecular recognition of apoAequorin for coelenterazine using chemical probes, the chiral deaza-analogs of (S)- and (R)-deaza-CTZ (daCTZ) for coelenterazine and of (S)-2- and (R)-2-hydroxymethyl-deaza-CTZ (HM-daCTZ) for 2-peroxycoelenterazine were efficiently prepared by the improvement method. The chiral deaza-analogs of (S)-daCTZ and (S)-HM-daCTZ selectively inhibited the regeneration step to aequorin by binding the catalytic site of coelenterazine in the apoAequorin molecule. The crystal structures of the apoAequorin complexes with (S)-daCTZ and (S)-HM-daCTZ were determined, suggesting that the hydroxy moiety at the C6-hydroxyphenyl group and the carbonyl moiety of the imidazopyrazinone ring in coelenterazine are essential to bind the apoAequorin molecule through hydrogen bonding. Therefore, the chiral deaza-analogs of coelenterazine can be used as a probe to study the interaction between coelenterazine and the related proteins including photoprotein, luciferase, and coelenterazine-binding protein.

Identifiants

pubmed: 34115795
doi: 10.1371/journal.pone.0251743
pii: PONE-D-21-10693
pmc: PMC8195370
doi:

Substances chimiques

Apoproteins 0
Recombinant Proteins 0
apoaequorin 0
Aequorin 50934-79-7
Calcium SY7Q814VUP

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

e0251743

Déclaration de conflit d'intérêts

The authors have declared that no competing interests exist.

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Auteurs

Satoshi Inouye (S)

Yokohama Research Center, JNC Co., Yokohama, Japan.

Yuto Sumida (Y)

Laboratory for Chemical Biology, RIKEN Center for Biosystems Dynamics Research (BDR), Kobe, Japan.

Yuri Tomabechi (Y)

Laboratory for Protein Functional and Structural Biology, RIKEN Center for Biosystems Dynamics Research (BDR), Yokohama, Japan.

Jumpei Taguchi (J)

Laboratory of Chemical Bioscience, Institute of Biomaterials and Bioengineering, Tokyo Medical and Dental University, Tokyo, Japan.

Mikako Shirouzu (M)

Laboratory for Protein Functional and Structural Biology, RIKEN Center for Biosystems Dynamics Research (BDR), Yokohama, Japan.

Takamitsu Hosoya (T)

Laboratory for Chemical Biology, RIKEN Center for Biosystems Dynamics Research (BDR), Kobe, Japan.
Laboratory of Chemical Bioscience, Institute of Biomaterials and Bioengineering, Tokyo Medical and Dental University, Tokyo, Japan.

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Classifications MeSH