Neuroendocrine Factors in Melanoma Pathogenesis.
melanoma
neurohormones
neuropeptides
neurotransmitters
stress
Journal
Cancers
ISSN: 2072-6694
Titre abrégé: Cancers (Basel)
Pays: Switzerland
ID NLM: 101526829
Informations de publication
Date de publication:
10 May 2021
10 May 2021
Historique:
received:
04
04
2021
revised:
03
05
2021
accepted:
05
05
2021
entrez:
2
6
2021
pubmed:
3
6
2021
medline:
3
6
2021
Statut:
epublish
Résumé
Melanoma is one of the most aggressive skin cancers with a sharp rise in incidence in the last decades, especially in young people. Recognized as a significant public health issue, melanoma is studied with increasing interest as new discoveries in molecular signaling and receptor modulation unlock innovative treatment options. Stress exposure is recognized as an important component in the immune-inflammatory interplay that can alter the progression of melanoma by regulating the release of neuroendocrine factors. Various neurotransmitters, such as catecholamines, glutamate, serotonin, or cannabinoids have also been assessed in experimental studies for their involvement in the biology of melanoma. Alpha-MSH and other neurohormones, as well as neuropeptides including substance P, CGRP, enkephalin, beta-endorphin, and even cellular and molecular agents (mast cells and nitric oxide, respectively), have all been implicated as potential factors in the development, growth, invasion, and dissemination of melanoma in a variety of in vitro and in vivo studies. In this review, we provide an overview of current evidence regarding the intricate effects of neuroendocrine factors in melanoma, including data reported in recent clinical trials, exploring the mechanisms involved, signaling pathways, and the recorded range of effects.
Identifiants
pubmed: 34068618
pii: cancers13092277
doi: 10.3390/cancers13092277
pmc: PMC8126040
pii:
doi:
Types de publication
Journal Article
Review
Langues
eng
Subventions
Organisme : Core Program
ID : PN 19.29.01.01
Organisme : Romanian Ministry of Research and Innovation, CCCDI-UEFISCDI
ID : 61PCCDI⁄2018 PN-III-P1-1.2-PCCDI-2017-0341
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