Nuclear and cytosolic pS727-STAT3 levels correlate with overall survival of patients affected by clear cell renal cell carcinoma (ccRCC).


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
26 03 2021
Historique:
received: 23 11 2020
accepted: 08 03 2021
entrez: 27 3 2021
pubmed: 28 3 2021
medline: 21 10 2021
Statut: epublish

Résumé

Clear cell renal cell carcinoma (ccRCC) is the most frequent and aggressive subtype of renal carcinoma. So far, the basis of its oncogenesis remains unclear resulting in a deficiency of usable and reliable biomarkers for its clinical management. Previously, we showed that nuclear expression of the signal transducer and activator of transcription 3 (STAT3), phosphorylated at its serine 727 (pS727), was inversely proportional to the overall survival of ccRCC patients. Therefore, in the present study, we validated the value of pS727-STAT3 as a clinically relevant biomarker in ccRCC. This work is a retrospective study on 82 ccRCC patients treated with nephrectomy and followed-up for 10 years. Immunohistochemical expression of pS727-STAT3 was analyzed on a tissue microarray and nuclear and cytosolic levels were correlated with clinical outcome of patients. Our results showed that pS727-STAT3 levels, whether in the nucleus (p = 0.002; 95% CI 1.004-1.026) or the cytosol (p = 0.040; 95% CI 1.003-1.042), significantly correlate with patients' survival in an independent-manner of clinicopathological features (Fuhrman grade, risk group, and tumor size). Moreover, we report that patients with high pS727-STAT3 levels who undergone adjuvant therapy exhibited a significant stabilization of the disease (~ 20 months), indicating that pS727-STAT3 can pinpoint a subset of patients susceptible to respond well to treatment. In summary, we demonstrated that high pS727-STAT3 levels (regardless of their cellular location) correlate with low overall survival of ccRCC patients, and we suggested the use of pS727-STAT3 as a prognostic biomarker to select patients for adjuvant treatment to increase their survival.

Identifiants

pubmed: 33772065
doi: 10.1038/s41598-021-86218-x
pii: 10.1038/s41598-021-86218-x
pmc: PMC7998019
doi:

Substances chimiques

Biomarkers, Tumor 0
Protein Kinase Inhibitors 0
STAT3 Transcription Factor 0
STAT3 protein, human 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

6957

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Auteurs

Jazmine Arévalo (J)

Renal Physiopathology CIBBIM-Nanomedicine, Vall d'Hebron Institut de Recerca (VHIR), Vall d'Hebron 119-129, 08035, Barcelona, Spain.

David Lorente (D)

Urology Department, Hospital Universitari Vall d'Hebron (HUVH), Barcelona, Spain.

Enrique Trilla (E)

Urology Department, Hospital Universitari Vall d'Hebron (HUVH), Barcelona, Spain.

María Teresa Salcedo (MT)

Pathology Department, Hospital Universitari Vall d'Hebron (HUVH), Barcelona, Spain.

Juan Morote (J)

Urology Department, Hospital Universitari Vall d'Hebron (HUVH), Barcelona, Spain.

Anna Meseguer (A)

Renal Physiopathology CIBBIM-Nanomedicine, Vall d'Hebron Institut de Recerca (VHIR), Vall d'Hebron 119-129, 08035, Barcelona, Spain. ana.meseguer@vhir.org.
Biochemistry and Molecular Biology Department, Universitat Autònoma de Barcelona (UAB), Barcelona, Spain. ana.meseguer@vhir.org.

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Classifications MeSH