COVID-19 and Oxidative Stress.
Journal
Biochemistry. Biokhimiia
ISSN: 1608-3040
Titre abrégé: Biochemistry (Mosc)
Pays: United States
ID NLM: 0376536
Informations de publication
Date de publication:
Dec 2020
Dec 2020
Historique:
entrez:
11
3
2021
pubmed:
12
3
2021
medline:
18
3
2021
Statut:
ppublish
Résumé
Pathogenesis of the novel coronavirus infection COVID-19 is the subject of active research around the world. COVID-19 caused by the SARS-CoV-2 is a complex disease in which interaction of the virus with target cells, action of the immune system and the body's systemic response to these events are closely intertwined. Many respiratory viral infections, including COVID-19, cause death of the infected cells, activation of innate immune response, and secretion of inflammatory cytokines. All these processes are associated with the development of oxidative stress, which makes an important contribution to pathogenesis of the viral infections. This review analyzes information on the oxidative stress associated with the infections caused by SARS-CoV-2 and other respiratory viruses. The review also focuses on involvement of the vascular endothelium in the COVID-19 pathogenesis.
Identifiants
pubmed: 33705292
pii: BCM85121816
doi: 10.1134/S0006297920120068
pmc: PMC7768996
doi:
Substances chimiques
Antioxidants
0
Cytokines
0
Reactive Oxygen Species
0
Angiotensin II
11128-99-7
Types de publication
Journal Article
Review
Langues
eng
Sous-ensembles de citation
IM
Pagination
1543-1553Commentaires et corrections
Type : CommentIn
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