Whole-genome sequencing analysis reveals the spread of a vanB-carrying transposon among different vancomycin-resistant Enterococcus faecium clinical isolates in a non-endemic setting.


Journal

The Journal of hospital infection
ISSN: 1532-2939
Titre abrégé: J Hosp Infect
Pays: England
ID NLM: 8007166

Informations de publication

Date de publication:
Apr 2021
Historique:
received: 23 07 2020
revised: 21 12 2020
accepted: 23 12 2020
pubmed: 8 1 2021
medline: 5 8 2021
entrez: 7 1 2021
Statut: ppublish

Résumé

Vancomycin-resistant enterococci (VRE), particularly Enterococcus faecium (VREfm), can cause serious nosocomial infections, and have been responsible for healthcare-associated outbreaks. Spreading of VREfm can occur both clonally and by the dissemination of mobile genetic elements. To report prospective analysis of whole-genome sequencing (WGS) data, including both core-genome multi-locus sequence typing (cgMLST) and transposon analysis, during a vanB VREfm outbreak. Screening for vanB-positive VREfm isolates was performed by real-time polymerase chain reaction (PCR) on an overnight enriched broth and, if positive, subculture was performed. vanB-positive VREfm isolates underwent WGS. Generated data were used for molecular typing that was performed by cgMLST using SeqSphere. For transposon characterization, sequence data were mapped against the reference sequence of transposon Tn1549 using CLC Genomics Workbench, or de-novo assemblies were used for BLASTN comparisons. In total, 1358 real-time PCRs were performed. Two hundred and fifty-one specimens from 207 patients tested positive on PCR for vanB, of which 13 specimens obtained from six patients were identified as vanB VREfm positive on culture. These six patients harboured seven unique isolates belonging to four cluster types: CT118 (N=2), CT2483 (N=3), CT2500 (N=1) and CT2501 (N=1). Transposon analysis revealed the presence of an identical vanB-carrying transposon in the isolates cultured from all six patients that could be linked based on epidemiological data. A vanB VREfm outbreak occurred in the study hospital, including six patients with isolates belonging to four cluster types. In-depth transposon analysis revealed that dissemination of transposon Tn1549 rather than clonal spread was the cause of the outbreak.

Sections du résumé

BACKGROUND BACKGROUND
Vancomycin-resistant enterococci (VRE), particularly Enterococcus faecium (VREfm), can cause serious nosocomial infections, and have been responsible for healthcare-associated outbreaks. Spreading of VREfm can occur both clonally and by the dissemination of mobile genetic elements.
AIM OBJECTIVE
To report prospective analysis of whole-genome sequencing (WGS) data, including both core-genome multi-locus sequence typing (cgMLST) and transposon analysis, during a vanB VREfm outbreak.
METHODS METHODS
Screening for vanB-positive VREfm isolates was performed by real-time polymerase chain reaction (PCR) on an overnight enriched broth and, if positive, subculture was performed. vanB-positive VREfm isolates underwent WGS. Generated data were used for molecular typing that was performed by cgMLST using SeqSphere. For transposon characterization, sequence data were mapped against the reference sequence of transposon Tn1549 using CLC Genomics Workbench, or de-novo assemblies were used for BLASTN comparisons.
RESULTS RESULTS
In total, 1358 real-time PCRs were performed. Two hundred and fifty-one specimens from 207 patients tested positive on PCR for vanB, of which 13 specimens obtained from six patients were identified as vanB VREfm positive on culture. These six patients harboured seven unique isolates belonging to four cluster types: CT118 (N=2), CT2483 (N=3), CT2500 (N=1) and CT2501 (N=1). Transposon analysis revealed the presence of an identical vanB-carrying transposon in the isolates cultured from all six patients that could be linked based on epidemiological data.
CONCLUSION CONCLUSIONS
A vanB VREfm outbreak occurred in the study hospital, including six patients with isolates belonging to four cluster types. In-depth transposon analysis revealed that dissemination of transposon Tn1549 rather than clonal spread was the cause of the outbreak.

Identifiants

pubmed: 33412230
pii: S0195-6701(20)30580-6
doi: 10.1016/j.jhin.2020.12.015
pii:
doi:

Substances chimiques

Bacterial Proteins 0
DNA Transposable Elements 0
Vancomycin 6Q205EH1VU

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

52-59

Informations de copyright

Copyright © 2021 The Healthcare Infection Society. Published by Elsevier Ltd. All rights reserved.

Auteurs

R H T Nijhuis (RHT)

Laboratory for Medical Microbiology and Medical Immunology, Meander Medical Centre, Amersfoort, The Netherlands. Electronic address: rht.nijhuis@meandermc.nl.

M A Chlebowicz-Fliss (MA)

University of Groningen, University Medical Centre Groningen, Department of Medical Microbiology and Infection Prevention, Groningen, The Netherlands.

A E Smilde (AE)

Department of Hygiene and Infection Prevention, Meander Medical Centre, Amersfoort, The Netherlands.

J W A Rossen (JWA)

University of Groningen, University Medical Centre Groningen, Department of Medical Microbiology and Infection Prevention, Groningen, The Netherlands; Department of Pathology, University of Utah School of Medicine, Salt Lake City, UT, USA.

A J L Weersink (AJL)

Laboratory for Medical Microbiology and Medical Immunology, Meander Medical Centre, Amersfoort, The Netherlands; Department of Hygiene and Infection Prevention, Meander Medical Centre, Amersfoort, The Netherlands.

A C M Gigengack-Baars (ACM)

Department of Hygiene and Infection Prevention, Meander Medical Centre, Amersfoort, The Netherlands.

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Classifications MeSH