Associations of Non-Alcoholic Beverages with Major Depressive Disorder History and Depressive Symptoms Clusters in a Sample of Overweight Adults.


Journal

Nutrients
ISSN: 2072-6643
Titre abrégé: Nutrients
Pays: Switzerland
ID NLM: 101521595

Informations de publication

Date de publication:
20 Oct 2020
Historique:
received: 09 09 2020
revised: 10 10 2020
accepted: 16 10 2020
entrez: 23 10 2020
pubmed: 24 10 2020
medline: 1 4 2021
Statut: epublish

Résumé

Meta-analysis of observational studies concluded that soft drinks may increase the risk of depression, while high consumption of coffee and tea may reduce the risk. Objectives were to explore the associations between the consumption of soft drinks, coffee or tea and: (1) a history of major depressive disorder (MDD) and (2) the severity of depressive symptoms clusters (mood, cognitive and somatic/vegetative symptoms). Cross-sectional and longitudinal analysis based on baseline and 12-month-follow-up data collected from four countries participating in the European MooDFOOD prevention trial. In total, 941 overweight adults with subsyndromal depressive symptoms aged 18 to 75 years were analyzed. History of MDD, depressive symptoms and beverages intake were assessed. Sugar-sweetened soft drinks were positively related to MDD history rates whereas soft drinks with non-nutritive sweeteners were inversely related for the high vs. low categories of intake. Longitudinal analysis showed no significant associations between beverages and mood, cognitive and somatic/vegetative clusters. Our findings point toward a relationship between soft drinks and past MDD diagnoses depending on how they are sweetened while we found no association with coffee and tea. No significant effects were found between any studied beverages and the depressive symptoms clusters in a sample of overweight adults.

Sections du résumé

BACKGROUND BACKGROUND
Meta-analysis of observational studies concluded that soft drinks may increase the risk of depression, while high consumption of coffee and tea may reduce the risk. Objectives were to explore the associations between the consumption of soft drinks, coffee or tea and: (1) a history of major depressive disorder (MDD) and (2) the severity of depressive symptoms clusters (mood, cognitive and somatic/vegetative symptoms).
METHODS METHODS
Cross-sectional and longitudinal analysis based on baseline and 12-month-follow-up data collected from four countries participating in the European MooDFOOD prevention trial. In total, 941 overweight adults with subsyndromal depressive symptoms aged 18 to 75 years were analyzed. History of MDD, depressive symptoms and beverages intake were assessed.
RESULTS RESULTS
Sugar-sweetened soft drinks were positively related to MDD history rates whereas soft drinks with non-nutritive sweeteners were inversely related for the high vs. low categories of intake. Longitudinal analysis showed no significant associations between beverages and mood, cognitive and somatic/vegetative clusters.
CONCLUSION CONCLUSIONS
Our findings point toward a relationship between soft drinks and past MDD diagnoses depending on how they are sweetened while we found no association with coffee and tea. No significant effects were found between any studied beverages and the depressive symptoms clusters in a sample of overweight adults.

Identifiants

pubmed: 33092067
pii: nu12103202
doi: 10.3390/nu12103202
pmc: PMC7589496
pii:
doi:

Substances chimiques

Coffee 0
Non-Nutritive Sweeteners 0
Tea 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : European Union FP7
ID : 613598

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Auteurs

M Ángeles Pérez-Ara (MÁ)

Institut Universitari d'Investigació en Ciències de la Salut, Idisba, Rediapp, University of Balearic Islands, 07122 Palma de Mallorca, Spain.

Margalida Gili (M)

Institut Universitari d'Investigació en Ciències de la Salut, Idisba, Rediapp, University of Balearic Islands, 07122 Palma de Mallorca, Spain.

Marjolein Visser (M)

Department of Health Sciences, Faculty of Science and the Amsterdam Public Health Research Institute, Vrije Universiteit Amsterdam, 1081 HV Amsterdam, The Netherlands.

Brenda W J H Penninx (BWJH)

Amsterdam UMC, Department of Psychiatry, Amsterdam Public Health Research Institute, Vrije Universiteit Amsterdam, de Boelelaan 1117, 1081 HV Amsterdam, The Netherlands.

Ingeborg A Brouwer (IA)

Department of Health Sciences, Faculty of Science and the Amsterdam Public Health Research Institute, Vrije Universiteit Amsterdam, 1081 HV Amsterdam, The Netherlands.

Ed Watkins (E)

Department of Psychology, University of Exeter, Exeter EX4 4QG, UK.

Matt Owens (M)

Department of Psychology, University of Exeter, Exeter EX4 4QG, UK.

Mauro García-Toro (M)

Institut Universitari d'Investigació en Ciències de la Salut, Idisba, Rediapp, University of Balearic Islands, 07122 Palma de Mallorca, Spain.

Ulrich Hegerl (U)

Department of Psychiatry, Psychosomatics, and Psychotherapy, Goethe-Universität Frankfurt, 60438 Frankfurt a.M., Germany.

Elisabeth Kohls (E)

Department of Psychiatry and Psychotherapy, Medical Faculty, University Leipzig, 04109 Leipzig, Germany.

Mariska Bot (M)

Amsterdam UMC, Department of Psychiatry, Amsterdam Public Health Research Institute, Vrije Universiteit Amsterdam, de Boelelaan 1117, 1081 HV Amsterdam, The Netherlands.

Miquel Roca (M)

Institut Universitari d'Investigació en Ciències de la Salut, Idisba, Rediapp, University of Balearic Islands, 07122 Palma de Mallorca, Spain.

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