Organized immune cell interactions within tumors sustain a productive T-cell response.


Journal

International immunology
ISSN: 1460-2377
Titre abrégé: Int Immunol
Pays: England
ID NLM: 8916182

Informations de publication

Date de publication:
01 01 2021
Historique:
received: 14 07 2020
accepted: 18 08 2020
pubmed: 23 8 2020
medline: 10 2 2022
entrez: 23 8 2020
Statut: ppublish

Résumé

Tumor-infiltrating CD8 T cells are associated with improved patient survival and response to immunotherapy in various cancers. Persistent antigen leads to CD8 T-cell exhaustion, where proliferation/self-renewal and killing are divided within distinct subsets of CD8 T cells in the tumor. CD8 T-cell responses in chronic antigen settings must be maintained for long periods of time, suggesting that mechanisms that regulate chronic CD8 T-cell responses may differ from those in acute settings. Currently, factors that regulate the maintenance of stem-like CD8 T cells in the tumor or their differentiation into terminally differentiated cells are unknown. In this review, we discuss the role of dendritic cells in the activation and differentiation of CD8 T-cell subsets within secondary lymphoid tissue and tumors. In addition, we examine changes in CD4 T-cell differentiation in response to chronic antigens and consider how subset-specific mechanisms could assist the stem-like and terminally differentiated CD8 T-cell subsets. Finally, we highlight how tumor-infiltrating CD4 T cells and dendritic cells interact with CD8 T cells within organized lymphoid-like areas in the tumor and propose a CD8 T-cell differentiation model that requires the collaboration of CD4 T cells and dendritic cells. These organized interactions coordinate the anti-tumor response and control disease progression by mechanisms that regulate CD8 T-cell differentiation, which permit the maintenance of an effective balance of stem-like and terminally differentiated CD8 T cells.

Identifiants

pubmed: 32827212
pii: 5895925
doi: 10.1093/intimm/dxaa057
pmc: PMC7771196
doi:

Substances chimiques

Antigens, Neoplasm 0

Types de publication

Journal Article Research Support, N.I.H., Extramural Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

27-37

Subventions

Organisme : NCI NIH HHS
ID : R00 CA197804
Pays : United States

Informations de copyright

© The Japanese Society for Immunology. 2020. All rights reserved. For permissions, please e-mail: journals.permissions@oup.com.

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Auteurs

Maria A Cardenas (MA)

Department of Urology, Emory University, Atlanta, GA, USA.

Nataliya Prokhnevska (N)

Department of Urology, Emory University, Atlanta, GA, USA.

Haydn T Kissick (HT)

Department of Urology, Emory University, Atlanta, GA, USA.
Department of Microbiology and Immunology, Emory University, Atlanta, GA, USA.
Emory Vaccine Centre, Atlanta, GA, USA.

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