Stress Echocardiography and Strain in Aortic Regurgitation (SESAR protocol): Left ventricular contractile reserve and myocardial work in asymptomatic patients with severe aortic regurgitation.


Journal

Echocardiography (Mount Kisco, N.Y.)
ISSN: 1540-8175
Titre abrégé: Echocardiography
Pays: United States
ID NLM: 8511187

Informations de publication

Date de publication:
08 2020
Historique:
received: 17 04 2020
revised: 08 06 2020
accepted: 12 07 2020
pubmed: 8 8 2020
medline: 24 6 2021
entrez: 8 8 2020
Statut: ppublish

Résumé

To analyze left ventricular (LV) myocardial deformation and contractile reserve (CR) in asymptomatic patients with severe aortic regurgitation (AR) at rest and during exercise, and their correlation with functional capacity. The natural history of chronic AR is characterized by a prolonged silent phase before onset of symptoms and overt LV dysfunction. Assessment of LV systolic function and contractile reserve has an important role in the decision-making of AR asymptomatic patients. Standard echo, lung ultrasound, and LV 2D speckle tracking strain were performed at rest and during exercise in asymptomatic patients with severe AR and in age- and sex-comparable healthy controls. 115 AR patients (male sex 58.2%; 52.3 ± 18.3 years) and 55 controls were enrolled. Baseline LV ejection fraction was comparable between the groups. Resting LV global longitudinal strain (GLS) and myocardial work efficiency (MWE) were significantly reduced in AR (GLS-15.8 ± 2.8 vs -21.4 ± 4.4; P < .001). Patients with AR and CR- showed reduced resting LV GLS and MWE and increased B-lines. MWE was closely related to peak effort watts, VO The lower resting values of LV GLS and MWE in severe AR asymptomatic patients suggest an early subclinical myocardial damage that seems to be closely associated with lower exercise capacity, greater pulmonary congestion, and blunted LV contractile reserve during stress.

Sections du résumé

OBJECTIVES
To analyze left ventricular (LV) myocardial deformation and contractile reserve (CR) in asymptomatic patients with severe aortic regurgitation (AR) at rest and during exercise, and their correlation with functional capacity.
BACKGROUND
The natural history of chronic AR is characterized by a prolonged silent phase before onset of symptoms and overt LV dysfunction. Assessment of LV systolic function and contractile reserve has an important role in the decision-making of AR asymptomatic patients.
METHODS
Standard echo, lung ultrasound, and LV 2D speckle tracking strain were performed at rest and during exercise in asymptomatic patients with severe AR and in age- and sex-comparable healthy controls.
RESULTS
115 AR patients (male sex 58.2%; 52.3 ± 18.3 years) and 55 controls were enrolled. Baseline LV ejection fraction was comparable between the groups. Resting LV global longitudinal strain (GLS) and myocardial work efficiency (MWE) were significantly reduced in AR (GLS-15.8 ± 2.8 vs -21.4 ± 4.4; P < .001). Patients with AR and CR- showed reduced resting LV GLS and MWE and increased B-lines. MWE was closely related to peak effort watts, VO
CONCLUSIONS
The lower resting values of LV GLS and MWE in severe AR asymptomatic patients suggest an early subclinical myocardial damage that seems to be closely associated with lower exercise capacity, greater pulmonary congestion, and blunted LV contractile reserve during stress.

Identifiants

pubmed: 32762102
doi: 10.1111/echo.14804
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1213-1221

Informations de copyright

© 2020 Wiley Periodicals LLC.

Références

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Auteurs

Antonello D'Andrea (A)

Unit of Cardiology, Department of Traslational Medical Sciences, Monaldi Hospital, University of Campania "Luigi Vanvitelli", Naples, Italy.
Unit of Cardiology and Intensive Coronary Care, Umberto I" Hospital, Nocera Inferiore, Italy.

Simona Sperlongano (S)

Unit of Cardiology, Department of Traslational Medical Sciences, Monaldi Hospital, University of Campania "Luigi Vanvitelli", Naples, Italy.

Tiziana Formisano (T)

Unit of Cardiology, Department of Traslational Medical Sciences, Monaldi Hospital, University of Campania "Luigi Vanvitelli", Naples, Italy.

Giampaolo Tocci (G)

Division of Cardiology, University of Perugia, Perugia, Italy.

Matteo Cameli (M)

Unit of Cardiology, Department of Medical Biotechnologies, University of Siena, Siena, Italy.

Maurizio Tusa (M)

IRCCS Polyclinic San Donato, Milan, Italy.

Giuseppina Novo (G)

Cardiology, Biomedical Department of Internal and Specialist Medicine, University of Palermo, Palermo, Italy.

Giovanni Corrado (G)

Valduce Hospital, Como, Italy.

Quirino Ciampi (Q)

Fatebenefratelli Hospital of Benevento, Benevento, Italy.

Rodolfo Citro (R)

Heart Department, AOU S. Giovanni e Ruggi, Salerno, Italy.

Eduardo Bossone (E)

UOC Cardiologia Riabilitativa, Cardarelli Hospital, Naples, Italy.

Maurizio Galderisi (M)

University of Naples Federico II, Naples, Italy.

Francesco Giallauria (F)

University of Naples Federico II, Naples, Italy.

Giuseppe Ambrosio (G)

Division of Cardiology, University of Perugia, Perugia, Italy.

Eugenio Picano (E)

Institute of Clinical Physiology, CNR, Pisa, Italy.

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