Effects of Strontium-Doped β-Tricalcium Scaffold on Longitudinal Nuclear Factor-Kappa Beta and Vascular Endothelial Growth Factor Receptor-2 Promoter Activities during Healing in a Murine Critical-Size Bone Defect Model.


Journal

International journal of molecular sciences
ISSN: 1422-0067
Titre abrégé: Int J Mol Sci
Pays: Switzerland
ID NLM: 101092791

Informations de publication

Date de publication:
01 May 2020
Historique:
received: 07 04 2020
revised: 29 04 2020
accepted: 29 04 2020
entrez: 7 5 2020
pubmed: 7 5 2020
medline: 7 2 2021
Statut: epublish

Résumé

It was hypothesized that strontium (Sr)-doped β-tricalcium phosphate (TCP)-based scaffolds have a positive effect on the regeneration of large bone defects (LBD). Readouts in our mice models were nuclear factor-kappa beta (NF-κB) activity and vascular endothelial growth factor receptor-2 (VEGFR-2) promoter activity during the healing process. A 2-mm critical-size femoral fracture was performed in transgenic NF-κB- and VEGFR-2-luciferase reporter mice. The fracture was filled with a 3D-printed β-TCP scaffold with or without Sr. A bioluminescence in-vivo imaging system was used to sequentially investigate NF-κB and VEGFR-2 expression for two months. After sacrifice, soft and osseous tissue formation in the fracture sites was histologically examined. NF-κB activity increased in the β-TCP + Sr group in the latter stage (day 40-60). VEGFR-2 activity increased in the + Sr group from days 0-15 but decreased and showed significantly less activity than the β-TCP and non-scaffold groups from days 40-60. The new bone formation and soft tissue formation in the + Sr group were significantly higher than in the β-TCP group, whereas the percentage of osseous tissue formation in the β-TCP group was significantly higher than in the β-TCP + Sr group. We analyzed longitudinal VEGFR-2 promoter activity and NF-κB activity profiles, as respective agents of angiogenesis and inflammation, during LBD healing. The extended inflammation phase and eventually more rapid resorption of scaffold caused by the addition of strontium accelerates temporary bridging of the fracture gaps. This finding has the potential to inform an improved treatment strategy for patients who suffer from osteoporosis.

Identifiants

pubmed: 32370039
pii: ijms21093208
doi: 10.3390/ijms21093208
pmc: PMC7246816
pii:
doi:

Substances chimiques

Bone Substitutes 0
Calcium Phosphates 0
NF-kappa B 0
Phosphatidylethanolamines 0
beta-tricalcium phosphate 0
dioleoyl phosphatidylethanolamine 2462-63-7
Vascular Endothelial Growth Factor Receptor-2 EC 2.7.10.1
Strontium YZS2RPE8LE

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

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Auteurs

Mersedeh Tohidnezhad (M)

Department of Anatomy and Cell Biology, RWTH Aachen University Hospital, 52074 Aachen, Germany.

Yusuke Kubo (Y)

Department of Anatomy and Cell Biology, RWTH Aachen University Hospital, 52074 Aachen, Germany.

Philipp Lichte (P)

Department of Trauma Surgery, RWTH Aachen University Hospital, 52074 Aachen, Germany.

Tobias Heigl (T)

Department of Anatomy and Cell Biology, RWTH Aachen University Hospital, 52074 Aachen, Germany.

Diana Roch (D)

Department of Anatomy and Cell Biology, RWTH Aachen University Hospital, 52074 Aachen, Germany.

Nazanin Barahmand Pour (N)

Department of Anatomy and Cell Biology, RWTH Aachen University Hospital, 52074 Aachen, Germany.

Christian Bergmann (C)

Department of Dental Materials and Biomaterials Research, RWTH Aachen University Hospital, 52074 Aachen, Germany.

Tolga Taha Sönmez (TT)

Department of Anatomy and Cell Biology, RWTH Aachen University Hospital, 52074 Aachen, Germany.
Department of Oral, Cranio-Maxillofacial and Facial Plastic Surgery, Hospital Karlsruhe of University Freiburg, 76133 Karlsruhe, Germany.

Jennifer Vanessa Phi Hock (JVP)

Department of Anatomy and Cell Biology, RWTH Aachen University Hospital, 52074 Aachen, Germany.

Athanassios Fragoulis (A)

Department of Anatomy and Cell Biology, RWTH Aachen University Hospital, 52074 Aachen, Germany.

Felix Gremse (F)

Institute for Experimental Molecular Imaging, RWTH Aachen University Hospital, 52074 Aachen, Germany.

Stefanie Rosenhain (S)

Institute for Experimental Molecular Imaging, RWTH Aachen University Hospital, 52074 Aachen, Germany.

Alexander Slowik (A)

Department of Neuroanatomy, RWTH Aachen University Hospital, 52074 Aachen, Germany.

Michaela Bienert (M)

Department of Pathology, RWTH Aachen University Hospital, 52074 Aachen, Germany.

Nisreen Kweider (N)

Department of Anatomy and Cell Biology, RWTH Aachen University Hospital, 52074 Aachen, Germany.

Christoph Jan Wruck (CJ)

Department of Anatomy and Cell Biology, RWTH Aachen University Hospital, 52074 Aachen, Germany.

Holger Jahr (H)

Department of Anatomy and Cell Biology, RWTH Aachen University Hospital, 52074 Aachen, Germany.
Department of Orthopedic Surgery, Maastricht UMC+, 6229 HX Maastricht, The Netherlands.

Frank Hildebrand (F)

Department of Trauma Surgery, RWTH Aachen University Hospital, 52074 Aachen, Germany.

Hans Christoph Pape (HC)

Department of Trauma Surgery, RWTH Aachen University Hospital, 52074 Aachen, Germany.
Department of Trauma, University Hospital and University of Zurich, 9GGX+JM Zürich, Switzerland.

Sabine Neuß (S)

Department of Pathology, RWTH Aachen University Hospital, 52074 Aachen, Germany.

Horst Fischer (H)

Department of Dental Materials and Biomaterials Research, RWTH Aachen University Hospital, 52074 Aachen, Germany.

Thomas Pufe (T)

Department of Anatomy and Cell Biology, RWTH Aachen University Hospital, 52074 Aachen, Germany.

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