Physical exercise augmented cognitive behaviour therapy for older adults with generalised anxiety disorder (PEXACOG): study protocol for a randomized controlled trial.

Brain-derived neurotrophic factor CBT Cognitive behavioural therapy Executive function GAD Generalised anxiety disorder Older adults Physical exercise RCT

Journal

Trials
ISSN: 1745-6215
Titre abrégé: Trials
Pays: England
ID NLM: 101263253

Informations de publication

Date de publication:
18 Mar 2019
Historique:
received: 25 06 2018
accepted: 28 02 2019
entrez: 20 3 2019
pubmed: 20 3 2019
medline: 6 8 2019
Statut: epublish

Résumé

Generalised anxiety disorder (GAD) is a frequent and severe anxiety disorder among older adults. GAD increases the risk of developing other disorders such as depression and coronary heart disease. Older adults with GAD exhibit a poorer response to cognitive behaviour therapy (CBT) compared to younger patients with GAD. The normal age-related cognitive decline can be a contributor to reduced treatment efficacy. One strategy for improving treatment efficacy is to combine CBT with adjunctive interventions targeted at improving cognitive functions. Physical exercise is a viable intervention in this regard. Increased levels of brain-derived neurotrophic factor may mediate improvement in cognitive function. The present study aims to investigate the proposed effects and mechanisms related to concomitant physical exercise. The sample comprises 70 participants aged 60-75 years, who have GAD. Exclusion criteria comprise substance abuse and unstable medication; inability to participate in physical exercise; and conditions which precludes GAD as primary diagnosis. The interventions are individual treatment in the outpatient clinic at the local psychiatric hospital, with two experimental arms: (1) CBT + physical exercise and (2) CBT + telephone calls. The primary outcome measure is symptom reduction on the Penn State Worry Questionnaire. Other measures include questionnaires, clinical interviews, physiological, biological and neuropsychological tests. A subset of 40 participants will undergo magnetic resonance imaging (MRI). After inclusion, participants undergo baseline testing, and are subsequently randomized to a treatment condition. Participants attend five sessions of the add-on treatment in the pre-treatment phase, and move on to interim testing. After interim testing, participants attend 10 sessions of CBT in parallel with continued add-on treatment. Participants are tested post-intervention within 2 weeks of completing treatment, with follow-up testing 6 and 12 months later. This study aims to develop better treatment for GAD in older adults. Enhancing treatment response will be valuable from both individual and societal perspectives, especially taking the aging of the general population into account. ClinicalTrials.gov, NCT02690441 . Registered on 24 February 2016.

Sections du résumé

BACKGROUND BACKGROUND
Generalised anxiety disorder (GAD) is a frequent and severe anxiety disorder among older adults. GAD increases the risk of developing other disorders such as depression and coronary heart disease. Older adults with GAD exhibit a poorer response to cognitive behaviour therapy (CBT) compared to younger patients with GAD. The normal age-related cognitive decline can be a contributor to reduced treatment efficacy. One strategy for improving treatment efficacy is to combine CBT with adjunctive interventions targeted at improving cognitive functions. Physical exercise is a viable intervention in this regard. Increased levels of brain-derived neurotrophic factor may mediate improvement in cognitive function. The present study aims to investigate the proposed effects and mechanisms related to concomitant physical exercise.
METHODS METHODS
The sample comprises 70 participants aged 60-75 years, who have GAD. Exclusion criteria comprise substance abuse and unstable medication; inability to participate in physical exercise; and conditions which precludes GAD as primary diagnosis. The interventions are individual treatment in the outpatient clinic at the local psychiatric hospital, with two experimental arms: (1) CBT + physical exercise and (2) CBT + telephone calls. The primary outcome measure is symptom reduction on the Penn State Worry Questionnaire. Other measures include questionnaires, clinical interviews, physiological, biological and neuropsychological tests. A subset of 40 participants will undergo magnetic resonance imaging (MRI). After inclusion, participants undergo baseline testing, and are subsequently randomized to a treatment condition. Participants attend five sessions of the add-on treatment in the pre-treatment phase, and move on to interim testing. After interim testing, participants attend 10 sessions of CBT in parallel with continued add-on treatment. Participants are tested post-intervention within 2 weeks of completing treatment, with follow-up testing 6 and 12 months later.
DISCUSSION CONCLUSIONS
This study aims to develop better treatment for GAD in older adults. Enhancing treatment response will be valuable from both individual and societal perspectives, especially taking the aging of the general population into account.
TRIAL REGISTRATION BACKGROUND
ClinicalTrials.gov, NCT02690441 . Registered on 24 February 2016.

Identifiants

pubmed: 30885256
doi: 10.1186/s13063-019-3268-9
pii: 10.1186/s13063-019-3268-9
pmc: PMC6423789
doi:

Banques de données

ClinicalTrials.gov
['NCT02690441']

Types de publication

Clinical Trial Protocol Journal Article

Langues

eng

Pagination

174

Commentaires et corrections

Type : ErratumIn

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Auteurs

Silje Haukenes Stavestrand (SH)

Faculty of Psychology, University of Bergen, Box 7800, NO-5020, Bergen, Norway. silje.h.stavestrand@uib.no.
Solli DPS, Osvegen 15, NO-5228, Nesttun, Norway. silje.h.stavestrand@uib.no.

Kristine Sirevåg (K)

Faculty of Psychology, University of Bergen, Box 7800, NO-5020, Bergen, Norway.
Solli DPS, Osvegen 15, NO-5228, Nesttun, Norway.

Inger Hilde Nordhus (IH)

Faculty of Psychology, University of Bergen, Box 7800, NO-5020, Bergen, Norway.
Faculty of Medicine, University of Oslo, Box 1078, Blindern, NO-0316, Oslo, Norway.

Trond Sjøbø (T)

Solli DPS, Osvegen 15, NO-5228, Nesttun, Norway.

Trygve Bruun Endal (TB)

Solli DPS, Osvegen 15, NO-5228, Nesttun, Norway.

Hans M Nordahl (HM)

Department of Mental Health, Norwegian University of Science and Technology, Box 8905, NO-7491, Trondheim, Norway.
St.Olavs Hospital HF, Nidaros DPS, Box 3250, Sluppen, NO-7006, Trondheim, Norway.

Karsten Specht (K)

Faculty of Psychology, University of Bergen, Box 7800, NO-5020, Bergen, Norway.

Åsa Hammar (Å)

Faculty of Psychology, University of Bergen, Box 7800, NO-5020, Bergen, Norway.

Anne Halmøy (A)

Faculty of Medicine, K.G. Jebsen Centre for Neuropsychiatric Disorders, University of Bergen, Box 7800, NO-5020, Bergen, Norway.
Kronstad DPS/Division of Psychiatry, Haukeland University Hospital, Box 1400, NO-5021, Bergen, Norway.

Egil W Martinsen (EW)

Faculty of Medicine, University of Oslo, Box 1078, Blindern, NO-0316, Oslo, Norway.
Division of Mental Health and Addiction, Oslo University Hospital, Oslo, Norway.

Eva Andersson (E)

The Swedish School of Sport and Health Sciences, GIH, Box 5626, SE-114 86, Stockholm, Sweden.

Helene Hjelmervik (H)

Faculty of Psychology, University of Bergen, Box 7800, NO-5020, Bergen, Norway.

Jan Mohlman (J)

Department of Psychology, William Paterson University, 300 Pompton Road, Wayne, NJ, 07470, USA.

Julian F Thayer (JF)

Department of Psychology, Ohio State University, 1835 Neil Avenue, Columbus, OH, 43210, USA.

Anders Hovland (A)

Faculty of Psychology, University of Bergen, Box 7800, NO-5020, Bergen, Norway.
Solli DPS, Osvegen 15, NO-5228, Nesttun, Norway.

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Classifications MeSH